Oxygen for cerebral infarction generally has no side effects, and oxygen is a very important therapeutic measure for large cerebral infarcts or brainstem infarcts. However, prolonged and highly concentrated oxygen therapy may have side effects.
After cerebral infarction, increased oxygen intake can elevate the oxygen concentration in the patient’s blood, which is conducive to the recovery of brain nerves. For cerebral nerves that are already necrotic, raising the oxygen concentration in the blood cannot restore the cells, but for the ischemic semi-dark zone around the infarcted area, raising the oxygen concentration in the blood can prevent the nerve cells at the ischemic semi-dark zone from continuing to degenerate and necrose, and is therefore conducive to the patient’s recovery.
When prolonged, high concentration oxygen therapy may be the side effects of oxygen therapy. Such as oxygen toxicity, pulmonary atelectasis, drying of respiratory secretions, respiratory depression and so on. Should follow the medical advice concentration of oxygen, encourage patients to do more deep breathing, to prevent secretions to block the airway, humidification and then inhalation of oxygen, for type 2 respiratory failure patients given low concentration, low flow continuous oxygen.
In addition to oxygen, patients with cerebral infarction should also be treated by correcting water-electrolyte disorders, using drugs such as mannitol to dehydrate to avoid cerebral edema, and using antiplatelet aggregation drugs such as aspirin.
If the diagnosis of cerebral infarction is confirmed, please follow the doctor’s instructions for treatment.