When the heart contracts, the anterior wall of the left ventricle strikes the chest wall in the precordial region early in systole, causing the corresponding part of the rib question tissue to pulsate outward, called apical pulsation. After myocardial infarction of the left ventricle, the entire myocardium of the ventricular wall is necrotic. In about 10-38% of cases, the necrotic myocardium is gradually replaced by fibrous scar tissue, forming a ventricular wall tumor. The thin layer of the ventricular wall in the lesioned area bulges outward, and the heart loses mobility or shows paradoxical motion during contraction. The evolution of coronary artery obstruction, myocardial infarction, myocardial fibrosis, and left ventricular wall tumors was well understood as early as 1881. Ventricular wall tumors in the left ventricle cause loss of myocardial contractility in the lesioned area and can produce reverse beats. The ventricular wall tumor expands outward during ventricular systole and retracts during diastole, resulting in a decrease in left ventricular ejection volume. Normal myocardial contractility is enhanced, tension is increased, and myocardial oxygen demand is increased. When the ventricular wall tumor volume exceeds the left ventricular end-diastolic volume by more than 15%, the left ventricular end-diastolic pressure increases. Left ventricular failure is progressively worsened by the impairment of left ventricular blood displacement. Once the thrombus is dislodged from the ventricular wall tumor, embolism of the body circulation can occur. The prognosis of left ventricular wall tumors is closely related to the extent of left ventricular myocardial involvement and the size of the tumor. Small tumors with limited LV myocardial involvement and asymptomatic or mild shortness of breath may survive for more than 10 years after acute myocardial infarction. If the lesion is large enough that the left ventricular systolic blood expulsion function is severely affected, the ejection fraction is significantly reduced, and the clinical presentation of congestive heart failure, the 5-year survival rate is reduced to about 10-20%.