1. Complicated cataract Complicated cataract is one of the common complications of anterior uveitis. The mechanism of its occurrence is not fully understood, but it is related to disorders of lens metabolism due to changes in the nature of atrial water during inflammation. Lens clouding secondary to posterior iris adhesions may be due to local hypoxia and accumulation of toxic metabolites due to changes in the direction of local atrial water flow. Enzymes produced during inflammation (e.g., phospholipase A and lysosomal enzymes) have been shown in animal studies to have damaging effects on the lens fiber membrane. Inflammatory products such as oxygen radicals have also been shown to cause lens damage in animal studies. Immune complexes deposited in the lens capsule membrane during inflammation can cause damage and exacerbate the inflammatory response near the lens through a complement cascade reaction. The glucocorticoids used to treat anterior uveitis can also induce cataracts. 2. Secondary glaucoma Anterior uveitis can cause glaucoma through a variety of mechanisms. In general, glaucoma caused by inflammatory debris and trabecular meshwork can be better treated with anti-inflammatory therapy; those with extensive posterior iris adhesions often require peripheral iridotomy or peripheral iridotomy; those with peripheral anterior iris adhesions, trabecular meshwork sclerosis, and iris redness can be treated with carbonic anhydrase inhibitors, timolol, and epinephrine, but those with poor results should be treated surgically; and those with glucocorticoid-induced glaucoma can be treated with a variety of other treatments. Glaucoma caused by corticosteroids can gradually return to normal after reducing or stopping the drug. The presence of banded keratoconus, which tends to occur in patients with chronic anterior uveitis, especially in patients with juvenile chronic arthritis associated with anterior uveitis, often indicates that the patient’s uveitis has lasted for a considerable period of time. 4. Low IOP and ocular atrophy Long-term chronic ciliary inflammation can lead to ciliary atrophy and reduced atrial fluid secretion, causing a decrease in IOP. In addition, the ciliary membrane pull on the ciliary body caused by inflammation and the resulting ciliary detachment can seriously affect atrial aqueous production, causing a significant decrease in IOP and, in severe cases, ocular atrophy.