Hypertensive cerebral hemorrhage, a kind of stroke, is usually caused by hypertension (about 60%) and is one of the most serious complications of hypertension; it is more common in patients aged 50-60 years old, more in the north than in the south, and is more common in the cold season, especially in the change of seasons, with a slightly higher incidence in men than in women. This disease has a high disability and mortality rate, and is a common disease, is one of the serious threats to our health in daily life, the onset of the disease in recent years has a younger trend, middle-aged and older friends should pay attention to. Symptoms: Sudden headache, vertigo, vomiting, hemiplegia, aphasia and even impaired consciousness are the main clinical manifestations. Most of them occur during strenuous exercise, mood swings, coughing and defecation, and also during rest and sleep. The onset of the disease is sudden and progresses rapidly, mostly with severe headache and vomiting, and soon with impaired consciousness and neurological deficits. The patient may be awake with a small amount of bleeding, but most of them have impaired consciousness, with drowsiness in mild cases and rapid coma in severe cases. Some patients with seizure onset or incontinence are the first symptoms. There is often contralateral hemiparesis and hemianesthesia, and dominant hemisphere hemorrhage may be associated with aphasia. If the disease progresses rapidly and brain herniation occurs, there will be increased muscle tone and positive pathological signs. The fundus may have retinal hemorrhage or optic disc edema, unequal pupils, bilateral narrowing or dilatation, deep inspiration, irregular rhythm, slow and strong pulse, elevated blood pressure, elevated body temperature, some patients may have acute gastrointestinal bleeding and vomiting of coffee-colored gastric contents. Depending on the site of hemorrhage (basal ganglia hemorrhage, thalamic hemorrhage, brainstem hemorrhage, cerebellar hemorrhage, ventricular hemorrhage), there may be different clinical features, such as: triple deviation, aphasia, ataxia, dysarthria, disturbance of vital signs, and in severe cases, herniation of the foramen magnum may occur, which is life-threatening. Disease treatment: The treatment is mainly conservative for those with less bleeding, but if the bleeding is large, surgery is required to remove the hematoma. Surgery: Surgery should be determined by a comprehensive evaluation of the patient’s general condition, age, state of consciousness, hematoma volume, bleeding site, and whether hydrocephalus is combined. In general, surgery is not required for patients with small amount of hemorrhage and consciousness, while surgery is not effective for patients who are deeply comatose, have dilated pupils bilaterally or even have unstable vital signs. For hemorrhage in the brain lobe and basal nucleus, craniotomy is feasible to remove the hematoma, and surgical treatment for thalamic hemorrhage is more cautious. Surgical methods 1.Craniotomy hematoma removal, according to the site of the hematoma design surgical access, direct vision to remove the hematoma, full decompression. 2.Borehole hematoma drainage, this method is not complete decompression, blind puncture may cause bleeding, should be used with caution. 3.Ventricular puncture and drainage, which is suitable for ventricular hemorrhage or posterior cranial fossa hemorrhage triggering obstructive hydrocephalus.