Lumbar synostosis accounts for a significant proportion of patients with low back pain. It is a syndrome that manifests itself due to degeneration of the intervertebral disc, rupture of the fibers, and protrusion of the nucleus pulposus, which irritates or compresses the nerve roots and cauda equina. The human spine is composed of 33 vertebrae: 7 cervical, 12 thoracic, 5 lumbar, 5 sacral and 4 caudal vertebrae. The cervical, thoracic and lumbar vertebrae are connected by intervertebral discs, while the sacral and caudal vertebrae, which have fused into one during development, do not have intervertebral discs. The intervertebral disc is composed of a tough peripheral fibrous ring and a jelly-like nucleus pulposus that is enclosed in the center. Under normal conditions, the intervertebral discs play both a connecting and stabilizing role for the spine, as well as a role in supporting spinal flexion and cushioning spinal pressure. In principle, every intervertebral disc has the potential to herniate, but especially between the 4th and 5th lumbar vertebrae and between the 5th lumbar vertebrae and the 1st sacral vertebrae are most likely to occur, accounting for about 90-96% of lumbar herniations. This is because the intervertebral discs between them are close to the lower end of the spine and usually have a large range of gravity and activity, so they have the most chance of damage and nucleus pulposus herniation. Why do you get lumbar synostosis? There are two main factors, one is ageing and the other is trauma. As we grow older, the water content of the main components of the disc, namely the annulus fibrosus and the nucleus pulposus, gradually decreases, the tension of the nucleus pulposus decreases, the disc thins, the nucleus pulposus gradually loses its elasticity, and the disc structure relaxes. At this time, under some stress, the nucleus pulposus can jack up the annulus fibrosus, which is called “bulge” or “herniation”. If the disc is damaged or even ruptured, the annulus fibrosus becomes weak. In mild cases, the nucleus pulposus can “herniate”; in severe cases, the nucleus pulposus can extrude along the rupture of the annulus fibrosus, known as “herniation” or “prolapse”. Whether “herniated” or “prolapsed,” the disc may compress the nerves that travel in the spinal canal and cause the clinical symptoms described above. The occurrence of lumbar herniation is undoubtedly related to lumbar activities and ageing factors. However, the causes are not limited to lumbar spine trauma or high intensity lumbar weight bearing. In clinical practice, most patients with lumbar synostosis do not have an obvious history of injury, and many of them are even brain workers. In fact, chronic strain on the lumbar back and lack of muscle strength around the lumbar spine are the main accomplices of lumbar synostosis. In modern society, lifestyles have changed a lot, with meetings, computers, driving, and car rides. People are sitting more and more, but the work pace is accelerating, but people have less and less time to exercise. This “more” a “less”, making many people prematurely appear lumbar muscle strain and lumbar spine “aging”. This is the direct reason for the increase in the prevalence of lumbar synostosis and the direct reason for the “rejuvenation” of the disease.