The goal of treatment for chronic gastritis is to relieve symptoms and improve histological changes in the gastric mucosa.
Different doctors in the clinic will give different treatments, and patients will ask questions such as.
”XX also has gastritis, and she is treated with XX medication, while why don’t I treat with that medication?”
”XX is treated effectively with XX medication, but I am not, why is that?”
In fact, as we have mentioned many times in our previous articles, there are different types of chronic gastritis, with different symptoms and different types of pathology. Different treatments are given for different conditions.
First, the principles of treatment for those with H. pylori infection are.
According to the consensus opinion of the 2007 China Lushan Conference, the principles of Helicobacter pylori eradication treatment are.
1, must be eradicated are: H. pylori infection positive peptic ulcer, early gastric cancer after surgery, gastric MALT lymphoma, chronic gastritis with gastric mucosal atrophy, erosion.
2. Those supporting eradication are: H. pylori positive chronic gastritis with dyspeptic symptoms, planned long-term NSAID use, family history of gastric cancer, unexplained iron deficiency anemia, idiopathic thrombocytopenic purpura, other H. pylori-associated gastric diseases (such as lymphocytic gastritis, gastric hyperplastic polyps, hypertrophic gastritis), and personal request for treatment.
Second, for chronic gastritis with delayed gastric emptying as the main symptom treatment principles.
The occurrence of epigastric fullness, nausea and vomiting may be related to delayed gastric emptying. Gastrointestinal stimulants such as domperidone, trimebutine maleate, mosapride and etopride hydrochloride can improve the above symptoms.
Third, chronic gastritis with gastric mucosal erosion accompanied by acid reflux and epigastric pain are the main symptoms treatment principles.
Chronic gastritis with gastric mucosal erosion or/and symptoms such as acid reflux, heartburn and epigastric pain can be treated with antacids or acid suppressants, such as H2 receptor blockers and proton pump inhibitors, depending on the condition or severity of symptoms.
Fourth, for chronic gastritis with pyloric insufficiency leading to bile reflux treatment principles.
Pyloric insufficiency leads to bile reflux and weakens or destroys the function of gastric mucosal barrier. Digestive juices act on the gastric mucosa, producing lesions such as congestion, erosion, bleeding, and epithelial metaplasia. Gastrointestinal stimulants such as domperidone, trimebutine maleate, mosapride, and etopride hydrochloride may improve symptoms and may prevent or reduce bile reflux. Gastric mucosal protective agents, such as aluminum thioglycollate, Rebapate tablets, teprenone, gemfacine, and sodium ecarbate can improve the gastric mucosal barrier, reduce the destruction of the gastric mucosal barrier by bile reflux, and promote the healing of gastric mucosal erosion, but the effect on the improvement of symptoms is controversial. Magnesium aluminum carbonate can enhance the gastric mucosal barrier function and can bind bile acid, thereby reducing or eliminating the damage to the gastric mucosa caused by bile reflux.
V. Principles of treatment of chronic gastritis with depression or anxiety symptoms.
Patients with chronic gastritis with obvious psychiatric factors can be treated with antidepressants or anxiolytics, and psychological guidance as well as psychotherapy should be given.
What is the regression of chronic gastritis?
Because the majority of chronic occurrence is related to Helicobacter pylori infection, and it is rare for Helicobacter pylori to feel cleared by itself, chronic gastritis can persist.
If chronic gastritis is not treated promptly and correctly, chronic gastritis can persist, H. pylori-associated gastric sinusitis is prone to gastroduodenal ulcers, multifocal atrophy is prone to gastric ulcers, and atrophic gastritis with enterosis or heterogeneous hyperplasia has a significantly increased risk of gastric cancer.