Angina pectoris is a group of clinical syndromes of angina pectoris caused by insufficient coronary artery blood supply, which is a contradiction between coronary artery blood supply and myocardial blood demand, and the coronary artery blood flow cannot meet the metabolic needs of myocardium, causing acute and temporary ischemia and hypoxia of myocardium. So, what causes can lead to anterior chest pressure pain? The following is a brief introduction: The direct cause of angina is the absolute or relative insufficiency of myocardial blood supply, therefore, various factors that reduce myocardial blood (blood oxygen) supply (such as intravascular thrombosis, vascular spasm) and increase oxygen consumption (such as exercise, increased heart rate) can induce angina. Inadequate myocardial blood supply mainly stems from coronary artery disease. Sometimes, other types of heart disease or uncontrolled hypertension can also cause angina pectoris. If fat continues to be deposited in the blood vessels, plaque can form. If the plaque occurs in the coronary arteries, it causes them to narrow, further reducing their blood supply to the heart muscle, and coronary heart disease is formed. The process of fat deposition in coronary arteries gradually forming plaques is called coronary atherosclerosis. Some plaques are harder and more stable, leading to narrowing and hardening of the coronary arteries themselves. Other plaques are softer and can easily break up to form blood clots. The accumulation of such plaque in the coronary artery walls can cause angina in two ways: the narrowing of the lumen at a fixed location in the coronary artery, which in turn leads to a significant reduction in blood flow through it; and the formation of blood clots that partially or completely block the coronary artery. It is often triggered by physical exertion, emotional stress, a full meal, shock, and cold. Typical angina attacks often occur under similar labor conditions or, in severe cases, while eating, dressing, defecating, or resting, with the pain occurring at the time of labor or excitement rather than after a day or bout of exertion. The onset of angina in the quiet state is the result of coronary artery spasm. The mechanism of pain onset in myocardial ischemia may be the result of stimulation of afferent nerve endings in the heart by certain products of myocardial anaerobic metabolism (e.g., acids such as lactate and pyruvate or peptides similar to kinins) and often propagates to superficial cutaneous nerves in the same spinal cord segment, causing painful radiation.