Ear frostbite, also known as auricular frostbite, is a condition in which the skin of the auricle becomes red, swollen, itchy, hot, and always wants to be rubbed with the hand, and gradually feels tingling, sometimes with shiny skin and blisters. In severe cases of auricular frostbite, the skin breaks down and ulcers can occur, and the auricle becomes yellow and crusty. Dry necrosis can occur in the auricle due to long-term ischemia and hypoxia, and it is not uncommon to see “frozen ears”. This can lead to lifelong ear loss and deformity. In the cold winter months, many people suffer from auricular frostbite. The skin of the auricle becomes red and swollen, itchy and hot, and you always want to rub it with your hands, gradually feeling the stinging pain, and sometimes the skin shines and blisters appear. In severe cases of auricular frostbite, the skin breaks down and ulcers can occur, and the auricle becomes yellow and crusty. Dry necrosis can occur in the auricle due to long-term ischemia and hypoxia, and it is not uncommon to see “frozen ears”. This can lead to a lifelong ear deformity and disfigurement. The reason why the ear is most susceptible to frostbite is closely related to the structure of the ear. In addition to the fatty tissue in the earlobe, the rest of the ear is covered with thin skin and cartilage, and the blood vessels inside are very thin, so the insulation capacity is extremely poor. Especially in winter, the ear is stimulated by the cold climate, the blood supply of the ear blood vessels will be less than other parts of the body, the end blood circulation is impaired, the Qi and blood do not run smoothly, and thus frostbite is likely to occur. The ear is exposed to the outside, so the ear suddenly changes from a warm environment to a cold environment and does not adapt, so it is most likely to freeze. After frostbite, the epidermis and dermis become edematous, the blood vessels become congested, and red thrombus formation is visible, followed by intimal hyperplasia and narrowing of the lumen. Skin attachments are atrophied or degenerated. Adipose tissue shows crystallization and necrosis, sometimes with free and intracellular fat droplets in the blood vessels (a unique feature of frostbite). As the degree of frostbite increases, the degree of tissue cell degeneration and necrosis also becomes more severe, which may manifest as dry or wet gangrene histopathological changes.