The clinical diagnosis of LVNC is mainly made by non-invasive examinations: transthoracic UCG and MRI, but the diagnostic criteria of these two methods are still highly controversial. Transthoracic echocardiography, due to its easy identification, wide application and low cost, is the most commonly used diagnostic method. Ultrasound diagnostic methods currently mainly use the four diagnostic criteria proposed by Jenni et al [8] 2001, with the ratio of end-systolic non-compaction ( non-compaction , NC) to dense myocardial layer ( compaction, C) greater than 2 as the main diagnosis. However, the diagnostic criteria for NC/C vary between 2:1 and 3:1 and have not been uniformly reported because of the different judgments and opinions between ultrasonographers regarding the normal variant and LVNC. The difficulty in making a definitive diagnosis using NC/C or myocardial trabecular thickness imaging is mainly due to the variation in location, density, and morphology of myocardial trabeculae.Punn and Silverman et al. retrospectively analyzed patients with LVNC using the 16-segment method of the American AHA and American Society of Ultrasonography and found that LVEF values were inversely related to the number of segments involved in non-dense myocardium. That is, the greater the extent of involvement, the worse the cardiac function. The prognosis (death and heart transplantation) was more pronounced in younger age groups, especially between 0 and 3 years of age, the more extensive the involvement. With the development of ultrasound techniques such as strain, strain rate and speckle tracking have been used to study LVNC, and although some investigators have done statistical analysis using NC/C, there is still a lack of a gold standard, and it is because of the lack of agreement in diagnosis that LVNC has been overdiagnosed or underdiagnosed. The use of cardiac MRI for the diagnosis of LVNC in both children and adults is increasing. The diagnostic criteria for LVNC on cardiac MRI are end-diastolic NC/C > 2.3, but the same controversy exists as for LVNC on echocardiography, and Jacquier et al. have suggested that a myocardial trabecular mass of 20% or more of the total LV myocardial mass is the diagnostic criterion for LVN C. Radiologists believe that the use of MRI myocardial 17 anatomical segment analysis provides a clearer identification of the myocardial trabecular layer and that MRI provides more information on myocardial fibrosis and delayed gadolinium visualization. It is important to note that thinning of the dense myocardial layer in the apical region of LVNC on cardiac MRI should be differentiated from apical ventricular wall tumors. The diagnostic study of LVNC by cardiac CT was started in 2001 and was considered to clearly demonstrate the structure of the non-dense myocardial layer of the left ventricle. since 2007 there has been an increasing trend, but CT diagnosis is not yet advocated because of the risk of tumors due to radiation exposure, especially in children and in patients with long term follow-up. The imaging diagnosis of LVNC at Cincinnati Children’s Medical Center in the United States uses several methods of data analysis. First, NC/C was measured by transthoracic echocardiography, and the traffic between the LV blood flow and the trabecular space was observed by color Doppler. The extent of non-dense myocardial layer involvement and the thickness of the dense myocardial layer were measured in detail and compared with normal values. Transthoracic short-axis views were used to find possible non-dense myocardium with stepwise rotational views. In addition, CMRI was used to assist in the diagnosis of LVNC, and gadolinium imaging was used to observe myocardial scarring, along with LV size, ventricular wall thickness, and ventricular systolic and diastolic function, while paying attention to the exclusion of congenital heart disease. Finally, the ventricle and myocardial trabeculae were carefully observed under cardiac dynamics.