Gout can be diagnosed through patient history, clinical manifestations and laboratory and imaging tests. Gout can be secondary to blood and kidney diseases, including acute and chronic leukemia, renal insufficiency, etc. In addition, a history of taking low-dose aspirin, furosemide, hydrochlorothiazide, etc., and relatives with gout or patients with hyperuricemia are more likely to develop gout. Acute attacks of gout can be characterized by severe joint redness, swelling, pain, increased local skin temperature, and difficulty moving. Laboratory tests for acute attacks of gout may show elevated white blood cell counts, elevated blood uric acid and uric acid, and arthropathy or urate crystal deposition may be seen on X-rays, joint ultrasound, CT, and other imaging tests. It is recommended that patients with suspected gout symptoms go to the rheumatology and immunology department of regular hospitals to further improve the relevant examinations to clarify the diagnosis.