There are many types of varicose veins, including varicose veins of the spermatic cord, varicose veins of the esophagogastric fundus, and varicose veins of the lower extremities, the most common of which are varicose veins of the lower extremities. There are many causes of varicose veins, but the fundamental problem is the increased pressure in the blood vessels. People are originally animals that walk on all fours and walk upright with their hands free. When upright, the pressure in the veins of the lower limbs is higher, and the anatomical structure of the human body determines that the walls of the veins far from the heart are weaker, and as a result, the weakest places bear the greatest pressure, and the veins are bound to dilate. There are valves in the veins, and the valves are one-way open “valves” that stop the backflow of blood from the veins. When varicose occurs, the valve function is also gradually lost. Increased venous pressure due to various factors (pregnancy, cokket syndrome) can cause excessive pressure on the valve, which gradually relaxes and disrupts the valve’s normal closing function. Lack of rhythmic movement of the lower extremities (prolonged standing in a fixed position) can also lead to increased venous pressure. Prolonged cold water irritation can cause imperceptible damage to the superficial veins and eventually, at some point, venous system disease. When venous blood flow is stagnant, the superficial veins gradually elongate, dilate, and twist, resulting in worm-like tortuosity, most commonly in the lower leg branches of the saphenous vein. Because the thickness and elasticity of the vessel walls are genetically related, this disease can occur in several families at the same time, mostly due to congenital weakness of the vein walls and poor valve structure, and many of these patients have liver and kidney cysts.