Convulsions in patients with cirrhotic ascites may be related to electrolyte disturbances such as low sodium and calcium caused by the disease. Cirrhosis is the terminal stage of liver disease, and the presence of ascites is a manifestation of the decompensated stage, which is a more severe stage of the disease. In this stage, patients will experience more complications, such as hypoproteinemia, anemia, and electrolyte disorders. Among them, a large amount of hepatic ascites will take away some of the sodium, calcium and other trace elements, resulting in electrolyte disorders. In long-term cirrhotic ascites and when the condition is particularly severe, hyponatremia and hypocalcemia will occur, which will cause gastrocnemius muscle spasms, twitching of hands and feet. If cirrhosis patients often have convulsions, they should inform their doctors in time for symptomatic treatment and management to avoid delaying their condition.