There is a correlation between H. pylori and gastric carcinogenesis, but there is no absolute causal relationship.
Current research shows that H. pylori can cause a variety of gastric diseases, including chronic gastritis, gastric ulcers, duodenal ulcers, and so on. Even with the aforementioned stomach diseases, it is not necessary to get stomach cancer. That is, although many people are infected with H. pylori, very few of those infected end up with gastric cancer.
The occurrence of gastric cancer is an extremely complex process, and H. pylori infection is only one of the risk factors. The toxic substances it secretes can damage the gastric mucosa, making it easier for various carcinogenic factors to directly alter and destroy gastric mucosal cells, which leads to gastric cancer. In other words, H. pylori is not the “killer” of gastric cancer, but it is the “accomplice” of gastric cancer because it destroys the protective barrier of gastric mucosa, which indirectly promotes the occurrence of gastric cancer.