What is secondary prevention of stroke?

  Secondary Prevention of Stroke
  A large study of stroke prevention was conducted in Europe
  Patients in the aspirin group were given aspirin 25 mg/dose twice a day;
  Patients in the pansentine group were given extended-release pansentine 200 mg/dose twice a day
  Patients in the combination M were given 25 mg/dose of aspirin + 200 mg/dose of extended-release pansentine twice a day and were compared with the placebo group.
  The results showed that.
  The aspirin/slow-release pansentine combination was more significant than the monotherapy in preventing recurrent stroke.
  Compared with the control group, the rate of secondary strokes.
  18% decrease in the aspirin group;
  16% in the pansentine group;
  In the combination group, there was a 37% decrease.
  Anticoagulation
  Not advocated for widespread use due to bleeding side effects and high laboratory requirements
  Use to prevent cerebral embolism is well established in certain cardiac diseases (rheumatic heart disease) because of the persistent embolic mechanism.
  Anticoagulation
  Identify the mechanism of stroke occurrence
  Appropriate use of anticoagulants such as warfarin
  Recommended INR: 2.0-3.0 safe and effective
  Identify atrial fibrillation induced cardiogenic embolism
  Aspirin plus anticoagulant
  Anticoagulant therapy
  Urokinase plus anticoagulant: AUST study
  Transarterial injection of urokinase via angiography in addition to oral anticoagulants reduces stroke recurrence and mortality more than oral anticoagulants alone
  Interventions for other heart diseases
  Treat the primary disease for its cause
  Myocardial infarction: maintenance of cardiac output: beta-blockers, ACEI, appropriate anticoagulants or antiplatelet agents
  Infective endocarditis: antibiotics
  Surgical intervention for carotid artery stenosis
  Looking for evidence of carotid stenosis
  Carotid ultrasound, MRA, TCD, DSA
  Intervention according to the degree of stenosis
  Mild stenosis (≤29%): conservative medical treatment
  Moderate stenosis (30%-69%): not sure
  Surgical intervention for carotid stenosis
  Severe stenosis (70%-99%): surgery
  Carotid endarterectomy can be performed repeatedly
  Other surgical procedures
  Carotid angioplasty
  Placement of carotid artery stents
  Intervention for homocysteinemia
  Homocysteinemia
  Plasma cysteine level above 16umol/l
  Interventions
  Folic acid 2mg, VitB12500ug, VitB625mg
  VISP and VITATOPS study
  Intervention TIA
  Patients with TIA are at risk of secondary stroke
  Interventions
  Removal of risk factors
  Timely application of anticoagulation therapy
  Administration of antiplatelet agents
  Interventions for post-stroke cognitive impairment
  Mostly after acute ischemic cerebrovascular disease
  Risk factors for cognitive impairment
  Advanced age, low education level, history of multiple cerebrovascular accidents, excessive
  Smoking, low arterial blood pressure, etc.
  Differentiation of post-stroke dementia from other related disorders
  Interventions
  Stabilization of blood pressure, neuroprotective agents, improvement of brain function and memory, etc.
  Pharmacological interventions
  Intervention for post-stroke cognitive impairment
  Aphasia intervention
  PASS II study
  Piracetam 12g/d×4weeks
  4.8g/d×3 months
  Intervention for post-stroke depression
  Incidence: 40%-50%
  High risk period: Initial 2 years after stroke
  Associated factors
  Older age at onset, high family burden, more infarct foci, lack of
  social contact, personality traits
  Interventions
  Drug therapy, psychotherapy, electroconvulsive therapy, etc.
  Management of lipids after stroke
  Increased risk of recurrence with plasma cholesterol >240 mg/dl
  Active monitoring of lipid levels
  Dietary control, physical exercise, pharmacological intervention (statins)
  SPARCL study
  Evaluate the protective effect of statin 80mg/d (ongoing study no results yet)
  Management of post-stroke glucose
  Although diabetes is considered an independent risk factor for ischemic stroke, it has not been established whether strict glycemic control reduces the incidence of stroke.
  In type 2 diabetes, sulfonylurea and/or insulin therapy may improve microvascular complications but not macrovascular complications, such as stroke.
  Management of post-stroke glucose
  Monitoring of blood glucose levels is controversial
  Diabetes mellitus as an uncertain risk factor
  Increased risk of recurrence with blood glucose >140 mg/dl
  GIST program
  Infusion of polarizing fluid to maintain normoglycemia
  Glycosylated hemoglobin (HBAIc) not associated with stroke recurrence
  Risk factor interventions
  Smoking
  Risk of stroke reduced by 50 complete abstinence from smoking after quitting smoking, %.
  Alcohol consumption
  Limit the amount of alcohol consumed
  Obesity
  WHR as a precise defining indicator
  Reasonable diet, physical activity
  Risk factor interventions
  Different interventions for different age, gender and ethnic groups
  Health education
  Change sedentary lifestyle
  Regular physical exercise
  Regular checkups and reasonable exercise programs for high-risk patients
  Rehabilitation of stroke patients
  Develop a rehabilitation plan
  Set rehabilitation goals
  Begin rehabilitation after the patient’s vital signs have stabilized
  Acute phase: prevention of disability
  Recovery phase: comprehensive rehabilitation (Bobath method, Brunnstrom
  method, weight reduction bicycle method, functional electrical stimulation, biofeedback therapy)