The principles of blood pressure control in the acute phase of cerebral hemorrhage are: after the acute phase of cerebral hemorrhage, the occupying effect caused by cerebral hemorrhage will cause an increase in intracranial pressure, and in order to ensure the blood supply to the brain tissue and the response of cerebrovascular autoregulation, the increase in intracranial pressure at this moment will cause a reactive increase in blood pressure, and if the blood pressure increases, it will cause the risk of rebleeding. Therefore, blood pressure needs to be actively controlled, and when systolic blood pressure is ≥180 mmHg and diastolic blood pressure is ≥105 mmHg, it needs to be controlled with drugs. The common antihypertensive drugs used in the acute phase are nitroglycerin, sodium nitroprusside, captopril, nifedipine, etc. However, in the acute phase, sodium nitroprusside or nitroglycerin is mostly used for intravenous pumping, because the two drugs have a more obvious antihypertensive effect and have a better effect on lowering blood pressure than oral medication.