Acute onset cerebrovascular disease, also known as stroke, is one of the more common causes of disability and death in patients with diabetes, accounting for about 10-15% of deaths from diabetes. Acute cerebrovascular disease in diabetic patients can be called diabetic cerebrovascular disease, which is currently classified under the category of macrovascular complications of diabetes, including ischemic stroke (cerebral infarction and transient ischemic attack) and hemorrhagic stroke (cerebral hemorrhage and subarachnoid hemorrhage). At present, total ischemic cerebrovascular disease accounts for about 60% and hemorrhagic cerebrovascular disease accounts for about 40% in China, but in diabetic patients mainly the incidence of ischemic stroke increases significantly, and the incidence of cerebral infarction is about four times higher than that in non-diabetic patients. The incidence of stroke in diabetic patients is higher in terms of mortality and disability, and recovery is slow. The onset of diabetic cerebrovascular disease is acute and sudden, with a rapid onset and rapid changes. Most of the attacks occur under the condition of poor control of blood glucose, blood lipids, blood viscosity or blood pressure, and under certain triggers. The main triggers include: hypoglycemia, emotional discomfort, excessive exertion, sudden climate change, obesity, pregnancy, overeating, heavy drinking, too little water, excessive sweating, persistent diarrhea, sudden change in body position and overexertion. These triggers are almost always associated with changes in blood viscosity and blood clotting status, and fluctuations in blood pressure interacting with pre-existing atherosclerosis. Once a diabetic patient develops symptoms such as unconsciousness or distorted mouth and eyes, unfavorable speech, abnormal sensation of limbs and hemiplegia, he should immediately undergo brain CT or MRI examination to clarify the diagnosis as soon as possible, measure blood glucose and electrolytes, etc., use insulin to correct hyperglycemia, and to avoid the aggravation of cerebral edema caused by too rapid a drop in blood glucose, blood glucose should be kept at a rate of 2.8-5.6 mmol/L (50-100 mg/dl) per hour. mg/dl), and the blood glucose changes should be observed while using dehydrating agents to avoid rebound. Hypertension at this time is related to stress and should not be controlled too low, but can be temporarily left untreated as long as there is no obvious hypertension. Anticoagulation and antiplatelet therapy can be given according to the patient’s coagulation function, and attention should be paid to the prevention and treatment of infection and nutritional support. Thrombolysis can be performed at an early stage for cerebral thrombosis in units with conditions. In the recovery period of the disease, combined Chinese and Western medicine rehabilitation treatment can be used to strengthen functional exercise. Diabetic cerebrovascular disease, like other complications of diabetes, is not only preventable but also preventable, and can be prevented from occurring or occurring later or to a lesser extent through good metabolic control and other measures. Therefore, taking active and effective precautions to avoid the above-mentioned triggers will help prevent diabetic cerebrovascular disease. Pay attention to frequent monitoring of blood glucose, blood lipids, blood pressure, blood viscosity and blood concentration of relevant inflammatory factors to keep them at normal or near normal levels, actively eliminate mood swings and maintain a stable state of mind, promptly replenish water when excessive sweating occurs, and seek medical attention when diarrhea and other uncomfortable symptoms occur to avoid worsening of symptoms leading to dehydration. When there is headache or numbness of limbs, go to hospital for examination to clarify the cause, especially when there is transient ischemic attack, we must pay attention to it and give systematic treatment to avoid further aggravation.