Patients with cerebral infarction generally cause a reactive increase in blood pressure. If patients develop hypotension, the following factors should be considered: 1. Inadequate volume, such as patients who are unable to eat because of impaired consciousness or difficulty swallowing, or insufficient intravenous rehydration, can cause a decrease in effective circulating blood volume, which can lead to hypotension. 2. Inappropriate use of blood pressure-lowering drugs, such as short-acting calcium antagonists like nifedipine, which tend to quickly lower blood pressure to a lower level, or excessive doses of antihypertensive drugs and the combined use of antihypertensive drugs can cause hypotension. If a patient with cerebral infarction develops hypotension, it tends to cause cerebral perfusion deficiency and infarction formation in the watershed area. Therefore, in the acute phase of cerebral infarction within 48 hours, unless the blood pressure exceeds 220/110 mmHg, antihypertensive treatment is generally not given, and after 48 hours, the blood pressure can be controlled to about 140 mmHg systolic and 90 mmHg diastolic, and should not be lowered too much.