The main criteria for determining hypertensive nephropathy are a history of hypertension, manifestations of renal damage, or can be diagnosed pathologically by renal puncture biopsy. Hypertensive renal damage is usually damage to the small arteries of the kidneys or renal parenchyma caused by primary hypertension. Renal disease caused by hypertension can be categorized into benign small artery nephrosclerosis and malignant small artery nephrosclerosis. 1. History of hypertension: benign small artery nephrosclerosis is caused by long-term uncontrolled benign hypertension, which lasts for 5~10 years; malignant small artery nephrosclerosis is caused by malignant hypertension, which progresses very rapidly. 2. Renal damage performance: benign small artery nephrosclerosis can appear nocturia, low specific gravity and low osmolality urine and other renal tubular concentration dysfunction, mild proteinuria, a small number of red blood cells and tubular, there may also be a decrease in creatinine clearance, blood creatinine increased. Malignant small artery nephrosclerosis can be seen in hematuria, large amount of proteinuria, tubular urine, etc., progressive deterioration of renal function, oliguria and so on. 3. Pathological manifestations: this disease mainly violates the small pre-glomerular arteries. Benign small artery nephrosclerosis leads to vitreous changes in the small glomerular arteries, and thickening of the intima-media of the interlobular arteries and the arterioles of the arcuate arteries. Malignant small artery nephrosclerosis can be seen in fibrinoid necrosis of the entry small arteries, interlobular arteries and arterioles of the arcuate arteries, and glomerular segmental necrosis and proliferative lesions can also occur. Patients with hypertensive nephropathy are advised to go to regular hospitals in time and standardize the treatment under the guidance of doctors.