Clinically, acromioclavicular joint dislocation accounts for about 12% of shoulder injuries. A significant number of patients miss the diagnosis because of “no significant abnormality” on x-ray, which leads to chronic acromioclavicular arthritis without timely and effective treatment. If a fall results in a dislocation of the acromioclavicular joint, the decision to treat the injury conservatively or surgically is often based on the severity of the injury. There are many ways to treat acromioclavicular dislocation, some of which have been eliminated, while some regions still use them for various reasons, such as internal fixation via acromioclavicular kyphosis, rostral lock screw fixation, acromioclavicular hook plate fixation, and rostral acromion displacement to reconstruct the rostral lock ligament (Dewer). If internal fixation of the acromioclavicular joint alone is performed without reconstruction of the rostral ligament, the rate of recurrent dislocation of the acromioclavicular joint after removal of the internal fixation is high. Transforaminal crestal pin internal fixation is prone to internal fixation migration, and reported sites of migration include: lung, liver, subclavian artery, and aorta, so it should be avoided. Rostral locking screw fixation may be associated with screw pullout and fracture, screw failure to enter the rostral process or penetration of the rostral base causing neurovascular injury. Reconstruction of the rostral locking ligament by displacement of the rostral eminence (Dewer) is a dynamic reconstruction. This operation is highly injurious, has a large local anatomical disruption, is prone to myocutaneous nerve injury, does not provide dynamic stability during rehabilitation, makes it difficult to maintain anatomical repositioning, increases the activity at the acromioclavicular joint, and causes joint instability and arthritis. The hooked plate fixation of the acromion may cause shoulder pain, acromion impingement, re-dislocation and other complications possibly, because the pointed hook may compress the supraspinatus tendon and the subacromial bursa and lead to sensation of distension and pain at the acromion and weakness of shoulder supination during active shoulder movement, which restricts the micro-movement of the acromioclavicular joint, thus leading to stress concentration at the medial part of the plate during functional exercise after reconstruction, and stress fractures have been reported in recent years. We currently use allograft tendon reconstruction of the rostral-clavicular ligament combined with wire anchor nailing to strengthen the treatment of acromioclavicular dislocations. The advantage of using allograft tendon is to avoid taking the tendon elsewhere in the patient’s body to replace the rostral collar ligament to be reconstructed, and the combined wire anchor nail fixation plays a double insurance role to ensure the stability of the acromioclavicular joint after tendon reconstruction, early postoperative rehabilitation without secondary surgery, and good long-term patient outcome.