Benign infantile convulsions with mild gastroenteritis (BICE), also known as mild gastroenteritis combined with convulsions, was first proposed by Japanese scholar Morioka in 1982. The etiology of BICE is not completely clear, but it is mostly thought to be related to viral infections, with rotavirus and norovirus being the most common. The pathogenesis of BICE is not yet clear, but some scholars believe that it has some commonality with febrile convulsions, and is related to the immature brain development of children, and under the influence of certain triggers (such as vomiting and diarrhea) the internal environment is affected, resulting in abnormal discharge of brain cells and convulsions. It has been reported that the level of cerebrospinal fluid NO is elevated in children with BICE, and it is presumed that NO and inflammatory factors have a role in the mechanism of convulsion occurrence. (1) the infant is previously healthy; (2) the season of onset is winter and spring; (3) the convulsions may not be febrile, and mild dehydration may be present, but there is no obvious hydroelectric acid-base disturbance; (4) the convulsions are mainly generalized tonic-clonic seizures, with many single episodes, but they may also occur in series, and may recur in one episode; (5) the cerebrospinal fluid examination is normal. (5) normal cerebrospinal fluid examination; (6) abnormal EEG during seizures, but normal interictal EEG; (7) no abnormal imaging changes in the brain; (8) good prognosis without recurrence and without development of epilepsy.