Gastric ulcers are not septic infections, but defects caused by the invasive action of gastric acid and pepsin on the mucosa.
Gastric ulcer is not caused by septic infection, but by damage factors and inadequate defense repair. Damage factors include the invasive effect of gastric acid and pepsin on the gastric mucosa, as well as drugs such as non-steroidal anti-inflammatory drugs and Helicobacter pylori infection, on the other hand, it is related to the decline in the defense of the gastric mucosal barrier.
Gastric ulcers are prevalent in the angle of the stomach or the side of the lesser curvature of the gastric sinus, and the typical manifestations include periodic epigastric pain, which can be manifested as vague pain, dull pain, hunger-like discomfort, etc., and often appear after eating.
The treatment of gastric ulcer includes general treatment and medication. General treatment includes maintaining a regular diet, abstaining from smoking and alcohol, drinking less strong tea, coffee and other stimulating beverages, and stopping unnecessary non-steroidal anti-inflammatory drugs.
Drug treatment includes proton pump inhibitors such as omeprazole and rabeprazole, gastric mucosal protectants such as magnesium aluminum carbonate, and amoxicillin and bismuth potassium citrate can be used if there is Helicobacter pylori infection.
Drugs need to be used under the guidance of a professional physician, and should not be used arbitrarily on their own.