Cirrhosis can not eat is mainly related to gastrointestinal stasis and edema, poor digestion and absorption, and intestinal flora dysbiosis.
Cirrhosis is the progression of various chronic diseases of the liver to a pathological stage characterized by chronic inflammation of the liver, diffuse fibrosis, pseudofollicularity, regenerative nodules, and intra- and extra-hepatic vascular proliferation, which is associated with hepatitis B virus infection, cholestasis, circulatory disorders, and parasitic infections.
Cirrhosis patients often have loss of appetite manifestation, mainly due to a series of pathological changes in the liver so that the circulation of the hepatic venous system is blocked or compressed, often causing portal hypertension, gastrointestinal venous reflux is blocked, resulting in the gastrointestinal wall of the stasis, edema, and at the same time affecting its digestive and absorptive functions, the patient appeared to have abdominal distension, loss of appetite symptoms.
The liver function of patients with cirrhosis is damaged, and its ability to secrete digestive juices is obviously reduced, which causes poor digestion and absorption to a certain extent. At the same time, due to the decline in the body’s immune function, changes in digestive enzymes and acidity, changes in the microenvironment in the intestinal tract, and dysbiosis of intestinal flora, it may cause loss of appetite.