Cerebral infarction refers to the obstruction of blood supply to the brain caused by various reasons, resulting in ischemia and hypoxic necrosis of brain tissue and corresponding neurological deficits. The most common causes of cerebral infarction: “three highs” hypertension, diabetes mellitus, hyperlipidemia. Cerebral infarction classification: atherosclerotic thrombosis; cerebral embolism; lacunar cerebral infarction; watershed cerebral infarction. Clinical manifestations of cerebral infarction: signs of limited neurological deficit after cerebral infarction. Determination of cerebral infarction FAST principle: auxiliary examination: cranial CT and MRI scan: the importance of cranial CT scan in the early stage of the onset of the disease is to exclude cerebral hemorrhage, but in the early stage of cerebral infarction CT has no abnormal findings, the infarcted area after 24~28 hours of the onset of the disease is obvious low-density changes, no occupying effect. MRI can be diagnosed 4 hours after the onset of the disease. Cerebrovascular examination: digital subtraction angiography DSA, CT or MR angiography can show the lesion site and nature of large arteries in the brain. It shows the site and extent of narrowing, occlusion or tortuosity of cerebral arteries. Transcranial Doppler examination (TCD) is a non-invasive method of examining cerebral hemodynamic changes, based on the flow rate and direction of blood flow, it can determine the presence of cerebral blood vessel stenosis and occlusion. The basic principles of treatment and prevention for different types of cerebral infarction are the same. Acute phase treatments should be selected based on the type of disease, the time window for treatment after onset, the severity of the disease, the underlying disease of the body and the complications, and individualized treatment plans should be implemented.