Benign prostatic hyperplasia is one of the most common benign diseases causing urinary disturbances in middle-aged and older men. It is a slowly progressive benign disease of the prostate whose symptoms worsen progressively as the patient ages. Initially it usually occurs after the age of 40, and by the age of 60 it is greater than 50% and up to 83% at the age of 80. The symptoms of prostatic hyperplasia are figuratively speaking, a knot at the bladder outlet, which is a faucet screwed down. A sense of incomplete urination, frequent urination, interruption of urination, urgency, thin urine lines, effortful urination and increased nocturia occur. The complications that arise are: urinary retention, hematuria, urinary tract infection, bladder stones, secondary hydronephrosis, combined with large diverticula of the bladder, inguinal hernia, hemorrhoids or prolapse. Mildly symptomatic patients can be treated with watchful waiting, including patient education, lifestyle coaching, and follow-up visits. The short-term goal of drug therapy is to relieve the patient’s lower urinary tract symptoms, and the long-term goal is to delay the clinical progression of the disease and prevent complications. Figuratively speaking, the bladder outlet is untied. Main drugs: alpha-blockers, 5α-reductase inhibitors, combination of the above two drugs, herbal and botanical preparations. Patients with severe symptoms consider surgical treatment modalities. Transurethral electrodesiccation of the prostate is now internationally recognized as the gold standard for the treatment of benign prostatic hyperplasia. In layman’s terms, it is a procedure that involves the removal of the prostate through a cystoscope. The advantages are less trauma (no incision), less bleeding, quicker recovery, shorter operation time, same efficacy as open surgery, wider indications than open surgery, and almost no complications. Figuratively speaking, the faucet is turned up.