The prostate gland is a male-specific organ, located in the pelvic floor, below the neck of the bladder, encircling the bladder opening and the urethra in the middle of the combination of the formation of the pipeline constitutes the urethra is a male-specific organ. Once the lesion, urinary difficulty becomes the first clinical manifestation. Prostate in men to the age of 45 or so began to appear two trends: a part of the prostate tissue gradually appeared atrophy; part of the gland volume gradually increased, the formation of the prostate hyperplasia. The pathological changes of hyperplasia generally begin to occur after the age of 40, and symptoms gradually appear after the age of 50. In recent years, due to changes in the living environment and lifestyle, prostate hyperplasia has gradually appeared a trend of lower age. At present, the etiology of prostate hyperplasia is still not very clear, there are three theories: (1) the role of sex hormones: in 1972, Willson firstly measured the DHT (dihydrotestosterone) content in the hyperplasia of prostate gland with the method of immunization is 2~3 times higher than the normal gland, and in the same gland, the first hyperplasia of the periurethral glands has a higher content of DHT than the other areas, and accordingly put forward the dihydrotestosterone theory, that the development of prostate hyperplasia is not only the development of prostate hyperplasia, it is also the cause of the disease, and it is the cause of the disease. It is believed that the occurrence of prostate hyperplasia is related to the accumulation of dihydrotestosterone in the gland. (2) prostate cells for embryonic reawakening: some studies have found that the initial pathological changes of prostate hyperplasia, that is, the formation of hyperplastic nodules only occurs in the region that accounts for 5%~10% of the prostate gland, McNeal according to the basic feature of embryonic development is the formation of new structures put forward the theory of embryonic reawakening of prostatic hyperplasia, that the formation of prostatic hyperplasia nodules is the formation of prostatic mesenchymal cells spontaneously transformed into embryonic embryonic embryonic embryonic embryonic embryonic embryonic embryonic embryonic embryonic reawakening of prostatic hyperplasia, that is, the formation of prostatic hyperplasia nodule is a prostatic interstitial cells in the process of growth. The formation of prostate hyperplasia nodules is the result of the spontaneous transformation of a particular prostate mesenchymal cell into an embryonic developmental state during the process. (3) Changes in the ratio of cell populations: It has been suggested that there is a strict hierarchy in the structure of the prostate gland: stem cells → amplifying cells → transition cells, in which an excessive number of stem cells can lead to an increase in the overall number of cells in the prostate gland, in which androgens stimulate the clonal proliferation of all the transition cells before leading to the formation of prostate hyperplasia. The 2011 European Annual Urological Congress by Israel’s chief scientist Dr. YIGAL GAT led a group of 63 cases of prostate hyperplasia patients in the study, through the theory of physics, came to a more certain conclusion, that is, the spermatic vein valve dysfunction, resulting in the concentration of testosterone is about 130 times the physiological concentration of testosterone through the testicles and the prostate gland through the venous reflux system of the testicles and the prostate gland from the testicles to reach the prostate gland, thereby This causes accelerated proliferation of prostate cells. This led to the invention of the “Gat Goren method” (a non-surgical, targeted interventional radiology method that seals off dysfunctional venous bypasses, as well as the many accompanying retroperitoneal collateral vessels that can cause the problem, in order to achieve therapeutic results in the treatment of prostate enlargement, by revealing the true cause of prostate enlargement development. They found that after 6-9 months of embolization, the volume of the pathologically enlarged prostate was significantly reduced by about 50%, with a parallel reduction in prostate symptoms. If this research result is further confirmed, it will open up a whole new approach to the future treatment of prostate hyperplasia, and dramatically reduce the pain and financial burden of patients.