It refers to the chronic partial or complete obstruction of the portal vein or intrahepatic branches of the portal vein, resulting in obstruction of portal blood flow and causing increased portal pressure. To reduce portal hypertension, varices may occur in the superficial abdominal wall veins located around the umbilicus after the formation of collateral circulation around the portal vein or recanalization of the obstruction. In hepatic portal hypertension, the main manifestations are portal hypertension and secondary rupture of esophagogastric fundic varices and/or associated portal hypertensive gastropathy. Patients may have recurrent vomiting of blood and tarry stools with mild to moderate splenomegaly and hypersplenism; therefore, the liver function of such patients is good, so ascites, jaundice and hepatic encephalopathy rarely occur. Occasionally, spongy degenerative collateral vessels may compress the superficial abdominal wall veins of the common bile duct around the umbilicus. For patients with recurrent upper gastrointestinal bleeding, mild or moderate splenomegaly, and basically normal liver function, the possibility of CTPV should be considered, and the diagnosis should be confirmed by ultrasound or color Doppler examination combined with portal venography. How to prevent abdominal wall vein anger? 1, avoid strain and excessive activity, ensure sufficient rest. Once dizziness, panic, sweating and other symptoms appear, you should rest in bed and gradually increase the amount of activity. 2, keep quiet, optimistic, stable emotions, avoid emotional tension, depression. 3, Quit smoking and alcohol. 4.Do not wear tight clothes, brush your teeth with a soft toothbrush, avoid constipation, sneezing, lifting heavy objects, etc. to reduce the risk factors of bleeding. The diagnosis can be confirmed by physical examination. Serum antinuclear antibodies, lupus anticoagulant, C protein, S protein and antithrombin III are normal, as are the international normalized ratio (INR) and activated partial thromboplastin time. However, the concentration of cardiac phospholipid antibodies IgM and IgG was high, and liver function tests were performed in addition to albumin, serum antinuclear antibodies, lupus anticoagulant, C protein, S protein and antithrombin, international normalized ratio (INR) and activated partial thromboplastin.