The favored sites of cerebral hemorrhage are, in order, the basal ganglia region, the cerebellum, the brainstem, and the lobes of the brain. Since the intracranial arteries have a middle myocyte layer and an outer connective tissue layer without an external elastic layer, they rupture during hypertension and atherosclerosis, causing vitreous degeneration and fibrinoid necrosis of small cerebral arteries. 1. The good part of hypertensive cerebral hemorrhage is the basal ganglia area, the bean artery supplying blood supply to the basal ganglia area occurs vertically in the middle cerebral artery, and in patients with hypertension, the bean artery is subjected to high-pressure blood for a long period of time, and vitrinoid degeneration and then rupture occurs, and cerebral hemorrhage leads to hemorrhage. 2. Hypertensive cerebral hemorrhage may also occur in the cerebellum and brainstem, the blood vessels in these areas are also some deep penetrating arteries, with age and fluctuations in blood pressure, vitreous lesions may occur, and then rupture and bleeding. 3. Cerebral hemorrhage due to cerebral amyloidosis and vascular malformation commonly occurs in the lobes of the brain. Basal nuclei, brainstem and cerebellum are rare. The most dangerous time for hematoma expansion is within 48 hours of the onset of the disease, and its incidence decreases as time progresses. Different causes of cerebral hemorrhage have different locations. After cerebral hemorrhage, symptoms such as headache and limb paralysis may occur, so it is important to seek medical attention in time and pay attention to controlling blood pressure in general.