Symptoms and dangers of hyperlipidemia

Many patients with hyperlipidemia often have no clinical symptoms. A few patients may develop fatty deposits with yellow tumors as the first manifestation, and the vast majority of patients are usually found through blood tests. With much elevated blood lipids, they may feel dizzy, bloated, weak, and drowsy. Long-term hyperlipidemia without active treatment may induce the formation of atherosclerosis in multiple systems, organs and parts of the body, for example, it may trigger lesions in the heart, brain, kidney and other blood vessels, resulting in chest pain, difficulty in breathing, insufficient blood supply to the brain and renal insufficiency. The common clinical hazards mainly include the following aspects: 1. Cardiovascular vascular lesions: if long-term hyperlipidemia is not taken seriously, patients may develop coronary atherosclerotic plaques, and if the plaques cause moderate or severe narrowing of the vascular cavity, patients will have corresponding angina symptoms, such as chest tightness, chest pain, dyspnea, palpitations, etc. The longer the time, the more serious the degree of atherosclerosis will be, serious patients will lead to target organ damage, and even clinical adverse events, the most common is high blood lipid induced coronary atherosclerotic heart disease, patients can appear angina, heart infarction symptoms, serious even endanger the lives of patients; 2, hypertension: after high blood lipid formation of atherosclerosis, myocardial function decreases, vascular artery spasm The two interact with each other, which will make the disease continue to develop; 3, cerebrovascular lesions: long-term hyperlipidemia may induce the hardening of cerebral arteries, and more serious cerebrovascular atherosclerosis may lead to symptoms such as dizziness, darkness, weakness and difficulty in breathing, and may also occur syncope and other paroxysmal cerebral blood supply deficiencies, and serious patients may develop cerebral infarction, etc.; 4, renal vascular lesions: from the perspective of the kidney, it may lead to multiple sclerosis of small arteries within the glomerulus, resulting in a decrease in the filtration rate of the glomerulus, causing renal insufficiency; 5, liver function damage: high blood lipids are not controlled for a long time, exceeding the function of liver synthesis, will develop into fatty liver, and further development will lead to cirrhosis, or even other malignant lesions, damaging liver function.