The preferred site of lacunar cerebral infarction is the basal ganglia region. Many patients with long-term hypertension, diabetes mellitus, and hyperlipidemia can lead to sclerosis of the small arteries of the brain, thickening of the vessel walls, and narrowing of the lumen, which can easily form small lacunar cerebral infarcts in the basal ganglia region. The lesions of lacunar cerebral infarction are small, perhaps around 2-15 mm in diameter, and may not cause patients to experience significant symptoms of neurological deficits. However, the basal ganglia region also contains important neurological nuclei, including, for example, the basal ganglia as well as the surrounding white matter and the region of the internal capsule. The internal capsule includes the anterior limb of the internal capsule, the knee of the internal capsule, and the posterior limb of the internal capsule. If a basal ganglia lacunar infarction occurs in the internal capsule area, it can lead to hemiparesis of the limb.