In brain infarction, the patient’s eyes cannot be opened. Consider that the brain infarction is blocked in the posterior circulation and in the brainstem, which is divided into the midbrain, cerebral bridge and medulla oblongata, and there are nuclei of the oculogyric nerve and the nucleus pulposus in the midbrain, of which the oculogyric nerve innervates the upper eye lift and the functions of internal, superior and inferior vision. When the nucleus of the locus coeruleus nerve is damaged, the patient will have symptoms such as inability to lift the upper eyelid, inability to internalize the eye, inability to lower the vision, and symptoms of midbrain infarction, such as dizziness and numbness. In case of brainstem bridge infarction, the brainstem bridge will have the spreading nerve passing through it, and if it is infarcted, the patient will have restricted abduction, and at the same time, the patient will also have the symptoms of brain bridge infarction. Therefore, brain infarction patients who cannot open their eyes consider blockage in the brainstem, which affects the motoneurotic nerve and the talocrural nerve that innervate the eyes, as well as some patients who have limited abduction, which affects the function of the spreading nerve.