It refers to the chronic partial or complete obstruction of portal vein branches in the portal part of the liver or within the liver, resulting in obstruction of portal blood flow and causing increased portal vein pressure. To reduce portal hypertension, varices may occur in the superficial abdominal wall veins located around the umbilicus after the formation of collateral circulation or obstruction around the portal vein. Etiology: In hepatic portal hypertension, the main manifestations are portal hypertension and secondary rupture of esophagogastric fundic varices and/or concomitant portal hypertensive gastropathy, patients may repeatedly vomit blood and tarry stools with mild to moderate splenomegaly and hypersplenism, so the liver function of such patients is good, so ascites, jaundice and hepatic encephalopathy rarely occur. Occasionally spongy degenerative collateral vessels may compress the superficial veins of the abdominal wall around the umbilicus of the common bile duct. For patients with recurrent upper gastrointestinal bleeding, mild or moderate splenomegaly and basically normal liver function, the possibility of CTPV should be thought of. Confirmation of the diagnosis requires ultrasound or color Doppler examination combined with portal venography.