I. Overview Ischemic myoclonus, also known as chronic congestive ischemic contracture, is a serious late complication of fracture and a serious consequence of osteofascial compartment syndrome. Due to insufficient blood supply to the upper and lower extremities or over-tightening of the bandage for more than a certain period of time, the limb muscles become ischemic and necrotic, eventually leading to mechanization and the formation of scar tissue, gradually contracting and forming a characteristic deformity, which is the final destination of most cardiovascular diseases and the most important It is the final destination of most cardiovascular diseases and the leading cause of death. The morbidity and mortality rates are not exactly known. All types of cardiac and macrovascular diseases can cause ischemic contracture. Ischemic contracture reflects the pumping dysfunction of the heart, that is, the diastolic insufficiency of the heart muscle. From the pathophysiological point of view, myocardial diastolic dysfunction can be broadly divided into two categories: primary myocardial damage and myocardial development from compensated to decompensated due to long-term cardiac overload: 1. Primary myocardial damage Ischemic myocardial damage, coronary artery disease, myocardial ischemia and/or myocardial infarction are among the most common causes of ischemic contracture. Myocarditis and cardiomyopathy various types of myocarditis and cardiomyopathy evenly and objectively cause ischemic contracture, with viral myocarditis and primary dilated cardiomyopathy being the most common. Myocardial metabolic disorders are most common with diabetic cardiomyopathy, others such as vitamin B1 deficiency and myocardial amyloid deformation are rare. 2, cardiac overload Pressure load (afterload) overload is seen in hypertension, aortic stenosis, pulmonary hypertension, pulmonary stenosis, etc., ventricular systolic ejection volume, persistent overload myocardium is bound to structural and functional changes and eventually loss of compensation, cardiac blood displacement decreases. Excessive volume load (preload) is seen in the following two conditions: ① heart valve closure insufficiency, blood regurgitation, such as aortic valve closure insufficiency, mitral valve closure insufficiency, etc.; ②, heart or arteriovenous shunt congenital cardiovascular disease such as septal defect, arteriovenous catheterization, etc.. In addition, diseases associated with increased systemic blood volume or increased circulating blood volume, such as chronic anemia and hyperthyroidism, are bound to increase the volume load of the heart. In the early stage of increased volume load, the ventricular cavity is compensated to expand to maintain normal cardiac output, but beyond a certain limit, there is a loss of compensatory performance. 3.Patients with underlying contracture, their ischemic contracture symptoms are often triggered by some factors that increase the cardiac load. The common causes of ischemic contracture are: (1) Infections Respiratory infections are the most common and important triggers. Infectious endocarditis is not uncommon as a trigger for ischemic contracture, but is easily missed because of its insidious onset. (2) Arrhythmia atrial tamponade is one of the most common arrhythmias in organic contracture and is the most important factor triggering ischemic contracture. Various other types of tachyarrhythmias as well as severe slow arrhythmias can induce ischemic contracture. (3) Increased blood volume such as excessive sodium intake, excessive and rapid intravenous fluid input, etc. (4) Excessive physical exertion or emotional excitement such as late pregnancy and delivery process, rage, etc. (5) Improper treatment such as improper discontinuation of digitalis drugs or antihypertensive drugs, etc. (6) Aggravation of existing contracture or complication of other diseases such as myocardial infarction in coronary heart disease, rheumatic activity in rheumatic heart valve disease, combined with hyperthyroidism or anemia, etc.