Smoking is the first to affect the function of the respiratory system. Domestic and international studies have shown that smoking is closely related to chronic bronchitis. The longer you smoke, the more you smoke, the higher the prevalence. After quitting smoking, the symptoms can be reduced or disappeared, and the condition can be alleviated or even cured. After inhaling smoke, parasympathetic excitability increases and bronchial contraction spasms; cilia movement of bronchial mucous membrane epithelial cells is inhibited; bronchial cup cells proliferate and mucus secretion increases. This is the reason why smokers usually have more phlegm than nonsmokers. Because of smoking, the bronchial mucosa becomes congested and edematous, and the local immune function is reduced, thus making smokers susceptible to lung diseases. A number of surveys have found that 80% to 90% of lung cancers in men are related to smoking, and 20% to 40% of lung cancers in women are related to smoking. Benzopyrene in tobacco is the main carcinogenic substance. Even passive smoking can easily cause lung cancer. When smoke passes through the smoker’s respiratory tract, there is also the possibility of cancer in the mouth and throat. In patients with atherosclerosis and coronary artery disease, the incidence of the disease and death rates increase 2 to 6 times in smokers compared to nonsmokers. It is also proportional to the number of cigarettes smoked per day. This is because smoking causes elevated lipid levels and changes in lipoprotein distribution, including an increased ratio of total cholesterol to HDL cholesterol, an important predictor of atherosclerotic cardiovascular disease. Studies have also found that these rates are increased in adolescents who are passive smokers, who are also at risk for developing atherosclerosis. Long-term smokers have a higher incidence of ulcer disease than nonsmokers, and smoking is more closely associated with gastric ulcers. Smokers have larger and slower healing ulcers, and even with effective medication, ulcers heal more slowly than nonsmokers. This is mainly because: nicotine in tobacco can mildly damage the gastric mucosa, and can exacerbate the effect of ethanol or anti-inflammatory and analgesic drugs on the gastric mucosa damage, but also the mucosa of prostaglandin E content reduction and thus lose its protective effect on the mucosa, and therefore prone to ulcer disease; long-term smoking can make the wall cell hyperplasia and increased secretion of gastric acid, producing self-digestion and ulcers; nicotine in tobacco can reduce the pyloric sphincter muscle Nicotine in tobacco can reduce the tension of the pyloric sphincter, making it easy for bile and duodenal fluid to flow back into the stomach, and inhibit the pancreatic secretion of bicarbonate, thus weakening the neutralizing ability of the duodenal cavity to gastric acid and predisposing to ulcers in the duodenal bulb. The incidence of bladder cancer in smokers is three times that of non-smokers. This is mainly because the chemicals in tobacco enter the bloodstream, are filtered by the kidneys, and are then excreted in the urine through the bladder. People who start smoking before the age of 20 are more likely to get bladder cancer than those who start smoking later. Smoking is one of the risk factors contributing to the prevalence of periodontal disease in the population. Researchers believe that the formation of gum plaque in smokers promotes the deposition of calculus, and local plaque irritation can also worsen periodontal disease. Tobacco contains a variety of harmful substances, and the chemical components of smoke also directly stimulate periodontal tissues, causing chronic damage to periodontal tissues. Smoking is not only harmful to oneself, but also to others. A survey of 1000 families found that more children under the age of 16 in smoking families suffered from respiratory disease than in non-smoking families. 33.5% of children under the age of 5 in non-smoking families had respiratory symptoms, while 44.5% of smoking families had respiratory symptoms. Passive smoking women who are using oral contraceptives increase the chance of heart attack and lower extremity venous thrombosis; and fetuses of passive smoking pregnant women are prone to premature birth and underweight, lowered immune function in infancy and childhood, and susceptibility to disease; statistically, the rate of teratogenicity in infants of passive smoking pregnant women is significantly higher. The Fukushima nuclear meltdown has been met with dismay, but smoking has not been taken seriously, but according to the Australian Times in September 2008, the radiation exposure of people who smoke a pack and a half of cigarettes a day is equivalent to 300 chest X-rays a year. Research published in the September 2008 issue of the American Journal of Public Health shows that smoking contains a dangerous radioactive substance, polonium-210, and that it is not yet possible to remove polonium-210 from cigarettes. Inhalation tests have shown that polonium-210 can cause lung cancer in animals. Having said all that, there may still be people who think that smoking has at least a relaxing benefit. That’s the wrong idea. The subjective feeling of every smoker is indeed comfort and relaxation. What is the reason? It is actually because nicotine stimulates the secretion of adrenaline in the body, and adrenaline significantly increases the body’s stress capacity, which leads to an increased ability to adapt to external stimuli, resulting in a subjective sense of relaxation. However, in reality, smoking causes a person’s blood pressure to rise, breathing to accelerate, and heart rate to accelerate, the opposite of what happens when a person is at rest. For the sake of your health and that of your family, we urge you to quit smoking as soon as possible.