Breathing gas for garlic odor is a symptom of organophosphorus pesticide poisoning, organophosphorus pesticide poisoning is an important sign due to the increased secretion of the glands, the poisoned person’s mouth and respiratory tract more with “garlic odor” secretions. So, what is the pathological reason for the emergence of garlic odor in the breath? The following is to analyze for you. Most of the human body efferent cholinergic nerves (including motor nerves, sympathetic, parasympathetic preganglionic fibers, parasympathetic nerves and part of the sympathetic postganglionic fibers) conduction, relying on the endings in the connection with the cell release of acetylcholine in order to innervate the effect of organ activity; some parts of the central nervous system, such as the cerebral cortex sensory-motor area, especially deep cortical pyramidal cells, caudate nucleus, thalamus, and other nerve cells Acetylcholine is also involved in the transmission of impulses between nerve cells. Cholinergic neurotransmission must bind to cholinergic receptors to produce effects. Cholinergic receptors are divided into muscarinic and nicotinic types; the former are found in effector organs such as cardiac muscle, smooth muscle, and glands innervated by postganglionic fibers of cholinergic nerves, and the latter are found in motor endplates of plant ganglia and skeletal muscle. Under normal conditions, the released acetylcholine is rapidly broken down by the acetylcholinesterase enzyme present in the tissues and loses its effect after completing its physiological function. When organophosphorus enters the human body, its phosphoryl group is tightly bound to the active part of the enzyme, forming phosphorylated cholinesterase and losing the ability to decompose acetylcholine, resulting in a large accumulation of acetylcholine in the body and inhibiting the vitality of the only acetylcholinesterase enzyme, so that the central nervous system and the cholinergic nerves are overexcited, and finally turn to inhibition and exhaustion to show a series of symptoms and signs: (1) some parasympathetic nerves and some sympathetic nerves ① some parasympathetic nerves and some postganglionic fibers of the cholinergic muscarinic receptor excitation, smooth muscle contraction, increased secretion of glands, pupil constriction, nausea, vomiting, abdominal pain, diarrhea, and other toxocara-like symptoms. ② Excitation of cholinergic nicotinic-type receptors at motor nerve and muscle junctions results in muscle fiber tremors or convulsions (spasms); in severe poisoning or late poisoning, it turns into nicotinic-like symptoms such as reduced muscle strength or muscle paralysis. (iii) In the central nervous system, excitation of interneuronal cholinergic receptors causes dysfunction, starting with excitatory symptoms such as headache, dizziness, restlessness and delirium, and in severe cases, speech disorders, coma and paralysis of the respiratory center. In the circulatory system, muscarinic symptoms such as slowed heart rate and decreased blood pressure, and nicotinic symptoms such as increased blood pressure and increased heart rate may occur.