(1) Internal capsule hemorrhage: It is the most common site of hemorrhage. The typical clinical manifestation is contralateral “triple hemiparesis” (hemiparesis, hemianesthesia and hemianopsia). The internal capsule hemorrhage is more extensive and the symptoms of nerve damage are more severe. However, if the hemorrhage is lateral to the internal capsule and mainly damages the external capsule, the clinical symptoms are less severe, and there is no consciousness disorder and hemiparesis is also mild, so the prognosis is better. (2) Thalamic hemorrhage: If the hemorrhage is on one side of the thalamus and the amount of hemorrhage is small, it shows light paralysis on the contralateral side and hemianesthesia on the contralateral side, especially the proprioceptive disorder is obvious. If the hemorrhage is large and the damaged area spreads to the contralateral thalamus and lower thalamus, vomiting of coffee-like material, frequent vomiting in the form of jets, polyuria, urinary sugar, limb paralysis, and double eye gaze to the tip of the nose are present. The disease is often critical and the prognosis is not good. (3) Lobar hemorrhage: also known as subcortical white matter hemorrhage, which can occur in any lobe of the brain. In addition to headache and vomiting, the clinical manifestations of hemorrhage in different lobes are also different. For example, frontal lobe hemorrhage may show psychiatric symptoms such as agitation, suspicion, contralateral hemiparesis, motor aphasia, etc.; parietal lobe hemorrhage may show contralateral sensory disorder; temporal lobe hemorrhage may show sensory aphasia, psychiatric symptoms, etc.; occipital lobe hemorrhage is most common with hemianopia. Lobar hemorrhage generally has slightly milder symptoms and a relatively good prognosis. (4) Pontocerebral hemorrhage: The pontocerebrum is a good site for brainstem hemorrhage. Early manifestation of lateral paresis at the site of the disease, the contralateral limb spread, called crossed paresis. This is the clinical characteristic of pontocerebral hemorrhage. (5) Cerebellar hemorrhage: If the hemorrhage is small, the clinical manifestations are often dizziness, followed by severe headache, frequent vomiting, unstable walking and unclear speech; if the hemorrhage is large and compresses the medulla oblongata vital center If the hemorrhage is large and compresses the vital center of the medulla oblongata, the patient may die suddenly. (6) Ventricular hemorrhage: generally divided into primary and secondary, primary ventricular hemorrhage is intracerebroventricular choroid plexus rupture hemorrhage, relatively rare. Secondary hemorrhage is due to the large amount of intracerebral hemorrhage, penetrating the brain parenchyma into the ventricles. Clinical manifestations are vomiting, excessive sweating, and purple or pale skin. One to two hours after onset, the patient falls into deep coma, high fever, limb paralysis or tonic convulsions, unstable blood pressure, and irregular breathing. The disease is mostly severe and the prognosis is poor.