Lacunar cerebral infarction is a type of cerebral infarction and is definitely a stroke without a doubt. Only lacunar cerebral infarction is a very small cerebral infarct lesion, usually less than 1.5 cm in diameter, accounting for about 20% of all cerebral infarcts that occur. Most of these infarcts occur in the deep basal ganglia region of the brain as well as in the brainstem and other areas. The arteries in these areas are mostly tiny arteries called deep penetrating branches, which are actually the terminal branches of the cerebral arteries, also known as end branches. Because of the limited blood supply of deep penetrating arteries, blockage of a single artery causes ischemic necrosis in only a small area of brain tissue, which is known as a lacunar infarct. Because the lesions are small and often located in relatively quiet areas of the brain, many cases cannot be recognized clinically during the acute onset. The deep basal ganglia region of the brain and the brainstem are important pathways for the travel of many nerve fiber bundles, which bridge the neural connections between the brain and the body. If lacunar cerebral infarction occurs in these pathways, it can cause blockage of certain nerve conduction and produce symptoms in terms of simple motor, sensory or speech impairment. Due to the small size of the lacunae, sometimes pure motor or sensory fibers are affected and pure motor hemiparesis occurs, or only hemianesthesia without hemiparesis occurs. It is important to emphasize that the “lacunar state” is due to a large number of multiple lacunar cerebral infarcts leading to a comprehensive impairment of brain function, followed by severe mental disorders (such as anxiety, depression, etc.), dementia, pseudobulbar palsy (described later), bilateral paralysis, incontinence, Parkinson’s syndrome and other symptoms, which often bring patients and their families a heavy burden in terms of material and psychological aspects. Thus, it appears that although lacunar cerebral infarction is small, the neurological deficits it causes can be severe. Although a single lacunar lesion rarely results in consequences as severe as coma, new foci of infarction can continue to appear as diffuse small cerebral artery degeneration has developed, forming multiple lacunar infarct foci. In the context of multiple stroke episodes, the accumulation and superimposition of this luminal brain damage is bound to cause more extensive brain dysfunction and can even lead to dementia, which is the most common cause of vascular dementia. Major countermeasures Effective treatment of hypertension and various types of cerebral atherosclerosis can reduce the incidence of lacunar cerebral infarction. However, it is not required to lower the blood pressure too low for those with lacunar status; a systolic blood pressure of 140-150 mmHg is better. Absolute smoking cessation, treatment of diabetes mellitus and dyslipidemia can reduce the occurrence of arteriopathy to some extent. Find a professional doctor to solve professional problems! Drugs such as oral aspirin and calcium antagonists can be taken, along with treatment of complications.