The patient’s choking on food and water is considered to be caused by brain problems. Specifically, it is divided into the following two aspects: first, it is considered to be caused by damage to the nuclei of the medulla oblongata of the brainstem, which is generally seen in the sequelae of cerebral infarction, and the pharyngeal nerves, vagus nerves and hypophyseal nerves of the patient are damaged, which results in choking and coughing, difficulty in swallowing, and hoarseness in drinking and eating; second, it should be considered that the patient has multiple lacunar cerebral infarctions, which results in damage to the cortical brainstem tracts and the corticospinal tracts of the brainstem and the corticospinal tracts of the brain, and causes central paralysis of the tongue, soft palate, pharynx, face and chewing muscles. The second aspect is to consider that the patient has multiple cavernous cerebral infarcts, causing bilateral cortical brainstem bundles and corticospinal bundles to be damaged, which causes the patient to develop central paralysis of the tongue, soft palate, pharynx, face and masticatory muscles, which is then manifested as choking on food and water. However, the patient may have multiple symptoms of lacunar cerebral infarction at the same time, and the patient may have slow reaction, slow gait, urinary and fecal incontinence, memory loss, and even cognitive decline.