High palatal arches are mostly seen in Marfan’s syndrome, mainly with elongated limbs, spider fingers (toes), arms extended flat with finger spacing greater than the length of the body, hands hanging down over the knees, upper body longer than the lower body. Long head deformity, narrow face, high palatal arch, large and low ears. Little subcutaneous fat, underdeveloped muscles, wrinkled skin on the chest, abdomen and arms. The muscle tone is low, with an inert body type. Ligaments, tendons and joint capsules are elongated and lax, and joints are hyperextended. Sometimes funnel chest, chicken chest, kyphosis, scoliosis, and spina bifida are seen. Cardiovascular lesions mainly invade the aorta, aortic valve and mitral valve and are the main cause of death. Since this disease is very characteristic, it can be described as a clear-cut disease. The disease can be demonstrated by a chain of genetic locus dominant inheritance in the family, as evidenced by increased urinary hydroxyproline excretion in the patient, and is an elastin fiber defect, also known as an abnormality of collagen metabolism. The primary defect of the disease is unknown. It has been suggested that the lateral association between elastin and collagen tissue peptide chains is impaired, i.e., a lysyl oxidase defect. It is also associated with acidic mucopolysaccharide deposition, increased sialic acid, hyaluronic acid accumulation, and poor formation or excessive destruction of chondroitin sulfate.