Xu, an elderly male, 73 years old, had a history of type 2 diabetes mellitus for 16 years and used insulin (eugenol 70/30, to control blood glucose for a long time. In January 1999, he was hospitalized in our hospital for cerebral infarction and was discharged with no sequelae and was able to take care of himself. Four days before his admission, he suddenly fell down while walking at home around 5:00 p.m. He was in a clear state of mind. On the fifth day, his family sent him to the hospital for treatment. On admission, the patient was drowsy, breathing steadily, mentally atrophied, with unfavorable movement of the left limb, unable to cooperate with the examination, and with slurred speech. Physical examination: emaciation, sometimes clear and sometimes confused, difficulty in tongue extension, muscle strength of the left limb grade 0, the right limb muscle strength grade 5. The left Bartholomew’s sign, Chaddock’s sign, Gordon’s sign and Oppenheim’s sign were all positive, muscle tone was significantly reduced, blood pressure was 160/70 mmHg, tongue was tender red, moss was cloudy and yellow, and pulse was stringent. Laboratory tests: FPG 11.1mmol/L, 2hPG 19.0mmol/L, HbA1c 8.7%. Cranial CT: right cerebellar infarction, right temporal and parietal old cerebral infarction. Traditional Chinese medicine diagnosis: 1. thirst (damp-heat interposition, 2. stroke Central visceral organs (wind-phlegm into the ligaments,; Western medicine diagnosis: type 2 diabetes mellitus, reentry (right cerebellar infarction). The incidence of cerebrovascular disease in diabetic patients is very high, about two times more than normal people of the same age, with diabetic heart disease and diabetic nephropathy as the three major causes of death in diabetic patients. And the occurrence of cerebrovascular disease in diabetic patients is more ischemic cerebrovascular disease. Hyperglycemia, hyperlipidemia and hypercoagulability are the risk factors for the onset and recurrence of cerebral infarction. The principles of cerebrovascular disease in diabetic patients are not well understood, but studies suggest that it is related to hyperinsulinemia, abnormal lipid metabolism, abnormal platelet function, endothelial damage of arterial vessels, hyperglycemia and abnormal metabolism of polyol pathways. Clinical typing includes several types of cerebral thrombosis, lacunar cerebral infarction, cerebral hemorrhage, and transient cerebral ischemia, etc. The diagnosis is not difficult by relying on typical clinical symptoms, CT, MRI and other radiological examination means. Diabetes combined with cerebral infarction belongs to the category of “thirst”, “stroke” and “vertigo” in Chinese medicine, which is often caused by factors such as worry and anger, poor diet and alcohol indulgence, resulting in the imbalance of yin and yang and the disorder of qi and blood. The onset of the disease is often caused by factors such as anxiety, anger, diet and alcoholism, resulting in the imbalance of yin and yang and the disruption of qi and blood. At the beginning of thirst, dry heat in the lung and stomach or damp heat in the spleen and stomach is the most common cause, and dryness injures fluids and heat scorches qi and fluids. Jin does not turn into gas, gas does not take in water, drink a urine, gradually become the next elimination, the disease from the lung and stomach and liver and kidney, both the blood and fluid depletion, liquid deficiency wind movement, take advantage of the orifice to steal luo, so the disease stroke. The deficiency of fluid and blood is of the same origin, the deficiency of fluid and blood is less, the fluid does not carry the blood and cannot travel smoothly through the meridians, so it can lead to blood stasis. Therefore, the essence is that deficiency causes stasis. The stasis of blood and blood vessels can be blocked, and the disease can develop, as stated in the Essentials of Treatment: “The three eliminations are prolonged, the essence and blood are deficient, or the eyes have no vision, or the hands and feet are paralyzed like wind disease, which is not wind.” The treatment plan after admission was as follows: mannitol and sodium beta heptaerythroside were used to dehydrate and reduce cerebral edema; cytarabine was used to improve energy metabolism and activate brain cells; blood pressure was monitored; insulin was used to strengthen blood glucose control; aspirin was used to antiplatelet aggregation and reduce blood viscosity; omeprazole was used to protect the gastric mucosa and avoid stress ulcers; the patient could not eat normally and was given enteral nutrition through a gastric tube after two days. At present, there are several aspects of Western medical treatment for cerebral infarction: first, controlling blood sugar; second, controlling blood pressure; in the acute stage, it is crucial to deal with the patient’s blood pressure to avoid the aggravation of cerebral ischemia; third, lowering intracranial pressure. Most cerebral infarctions occur with cerebral edema, but it is usually not a problem, especially in elderly people who already have varying degrees of brain atrophy can comply with less severe brain swelling. In young patients or large infarcts of the brain and cerebellar hemispheres, brain swelling reaches the most serious degree 2 to 5 days after the onset of the disease, and death can be caused by brain herniation due to increased intracranial pressure. Fourth, lower the body temperature. Lowering the body temperature can reduce the volume of experimental cerebral infarction, while increasing the body temperature will aggravate the scope of damage. A considerable number of patients have a history of infection before the onset of cerebral infarction, which is often complicated by pulmonary infection. V. Antithrombotic therapy. Anticoagulants and thrombolytic agents are mostly used, and anticoagulants are mainly used for progressive non-hemorrhagic infarction to limit the progress of infarction. They are contraindicated in patients with bleeding tendency, history of ulcer, severe hypertension, liver and kidney disorders and excessive age, and are not recommended for lacunar infarcts. Small doses of aspirin inhibit platelet production of thromboxane A2, which promotes platelet aggregation and vasoconstriction, and ticlopidine inhibits the binding of platelets and fibrinogen. After five days of treatment, the patient’s condition did not improve significantly and still showed drowsiness, mental atrophy, unfavorable movement of the left limb, inability to cooperate with the examination, and slurred speech. According to the condition at that time, herbal treatment was added. The patient was treated with blood clotting and brain-awakening sedative drops to remove blood stasis and open the body and wake up the brain; the Chinese herbal soup was gavaged twice a day, 100 ml each time. analysis of the evidence: the evidence of elimination is prolonged, the essence and blood is depleted day by day, the sympathetic yin transforms into wind and carries phlegm to take advantage of the orifice and steal the luo, so the mind is sometimes clear and sometimes obscure, the left side of the limb is hemiplegic, the coating is cloudy and yellow, the pulse is stringent. The treatment is to extinguish wind and eliminate phlegm, to awaken the brain and open the orifices. The patient was no longer drowsy, could eat slowly, no choking cough, normal answers, slow speech but clear spitting, left side muscle strength grade 1, negative pathological reflexes, and was discharged from the hospital. After discharge, he insisted on taking traditional Chinese medicine and western medicine such as insulin, mono and aspirin. After six months of follow-up, his speech had completely returned to normal, his left muscle strength had returned to level 4, he could walk on crutches and could take care of himself. This case belongs to the category of “thirst” and “stroke” in Chinese medicine. Huangdi Nei Jing? Suwen”, “internal seizure and syncope, the development of prickly heat. This is a case of internal seizure, which refers to the internal seizure of essence and blood, aphasia, and prickly heat, which is a movement disorder. The first evidence of elimination, the lung and stomach dry heat, the first injury to the lung and stomach fluids, followed by injury to the liver and kidney essence and blood, essence and blood is deficient, wind and yang excited over, with phlegm and turbidity take advantage of the orifice to steal the luo, and the disease stroke. In this case, the disease has been eliminated for 16 years, which is known as the depletion of liver and kidney essence and blood, Yin deficit in the lower part of the body, Yang hyperactivity in the upper part of the body, the wind of the sympathetic Yang, with phlegm and turbidity blocking the orifices of the machine, the gods can not stand by themselves, so the mind is sometimes clear and sometimes obscure, speech is bold, it is difficult to extend the tongue, the internal wind with phlegm and turbidity stealing the luo channel, the left side of the limbs are slow and vertical. The moss is cloudy and yellow, a sign of phlegm and turbidity staying in the body, and the pulse is stringent and strong, a sign of internal wind agitation. Therefore, the proposed treatment is to extinguish the wind and open the ligaments, and to cleanse phlegm and open the orifices as a preparation. After a long time of treatment, the illnesses receded, and the medicine was consistent with the evidence, so that the effect can be taken as quickly as this.