Renal infarction is similar to myocardial infarction in the sense that the artery supplying blood to the kidney is occluded. Whether it is caused by thrombosis, tumor compression, or vasospasm, or some trauma, as long as the kidney blood supply is interrupted or reduced, causing ischemic damage to the kidney. The kidney requires a large amount of blood circulation and is very sensitive to ischemia, especially the renal tubular cells, once the blood supply is reduced, there will be epithelial cell detachment and necrosis, resulting in acute deterioration of kidney function. Moreover, if renal infarction occurs, the back pain on the infarcted side will be very obvious, and at this time we can see that the local contrast agent cannot reach this part of the kidney by contrast therapy. At this time, when we check the ultrasound of both kidneys, the kidney on the infarcted side is larger than the opposite side, and early treatment of renal infarction is very important. After the interventional treatment, it can actively and effectively open up the perfusion of the kidney and prevent further damage to the kidney function. However, if the time is long, the kidney on this side of the obstruction will be permanently lost.