Prevention of cerebral infarction 1, pay attention to lifestyle: change the bad habits of smoking, alcoholism, staying up late, more meat and less food, sedentary, not exercise. Daily life requires more attention, less strain, regularity in living and living, do not overwork. Maintain moderate physical activity and exercise, while avoiding vomiting, diarrhea, heavy sweating and other conditions that lead to massive dehydration. Improve the diet structure, high fiber, high vitamin, low fat diet, and eat more vegetables, fruits, soy products, do not be overly full. Smoking and excessive alcohol consumption are both risk factors for cerebral infarction, so it is best to quit smoking and drink less alcohol. 2, mood regulation: maintain emotional stability, mood, avoid the storm, anger, worry and other mental stimulation. 3, treatment of primary diseases: hypertension makes the development of cerebral atherosclerosis accelerated and aggravated, diabetes patients complicating cerebrovascular disease is mostly ischemic. Patients need to actively receive treatment to control blood pressure and blood sugar. 4, regular review follow-up: patients who have suffered from cerebral infarction, it is best to go for a review once every six months, it is necessary to pay attention to the review of blood pressure, blood lipids, blood rheology and other indicators, early detection of problems and early resolution. The onset of cerebral infarction aura 1, dizziness, headache suddenly aggravated or from intermittent headache to continuous severe headache. It is generally believed that headache and dizziness are mostly the aura of ischemic cerebral infarction, while severe headache with nausea and vomiting is mostly the aura of hemorrhagic cerebral infarction. 2. Transient visual impairment, which is manifested as blurred vision or visual field defects and incomplete vision, mostly recovers on its own within one hour, and is an early forecasting signal of cerebral infarction. 3.Language and mental changes, such as difficulty in pronunciation, aphasia, difficulty in writing; sudden personality change, silence, indifferent expression or impatience, irritability, or transient judgment or intellectual impairment, drowsiness. 4, Other aura manifestations, such as nausea and vomiting, or blood pressure fluctuation with dizziness or tinnitus, unexplained recurrent nasal bleeding, often as a recent precursor of hypertensive cerebral hemorrhage. 5. Sleepiness and drowsiness, manifested by yawning, especially the response of hypoxia in the whistling center. As cerebral atherosclerosis worsens, the arterial lumen becomes narrower and cerebral ischemia worsens severely. 80% or so of people yawn frequently 5 to 10 days before the onset of ischemic cerebral infarction, so do not ignore this important alarm signal. 6.Somatosensory and motor abnormalities, such as episodic unilateral limb numbness or weakness, loss of hand grip on objects, unexplained fainting or falling, unilateral facial palsy, lasting less than 24 hours. 7, razor landing phenomenon, is the process of shaving, when the head turned to the side, suddenly feel the arm holding the razor weakness, razor landing, can be accompanied by slurred speech, but in 1-2 minutes or so completely back to normal. This is due to the fact that when the neck is turned, it aggravates the already sclerotic carotid artery stenosis, resulting in insufficient cranial blood supply and transient cerebral ischemia. It suggests that ischemic cerebral infarction may occur at any time. 8. Transient black haze refers to the sudden appearance of blackness in front of the eyes of a normal person, who cannot see objects, and then returns to normal in a few seconds or minutes, without nausea, dizziness, or any impairment of consciousness. This is caused by transient ischemia of the retina, suggesting intracranial hemodynamic changes or temporary blockage of retinal arteries by tiny blood clots, which is the earliest alarm signal of cerebrovascular disease.