Some of the brain infarcts will lead to cerebral hemorrhage, when the area of brain infarct is large, due to the ischemia of its own trophoid artery within the infarct lesion, resulting in the damage and necrosis of the arterial blood vessel wall, on the basis of which if the blood clot in the lumen of the vessel dissolves or its collateral circulation opens, etc., so that the blood flow of the already damaged vessel is restored, the blood will leak out from the broken vessel wall, thus triggering hemorrhagic cerebral infarction, which accounts for clinically all Hemorrhagic cerebral infarction accounts for about 20% of all patients with cerebral infarction in clinical practice. When the bleeding is large, cranial CT can make a clear diagnosis. In the case of microhemorrhage, magnetic sensitivity is relied upon to confirm the diagnosis. The treatment principles for hemorrhagic cerebral infarction are different from those for general cerebral infarction, as antiplatelet or anticoagulant drugs are not recommended in the acute phase, and antiplatelet and statin therapy is generally recommended for patients with hemorrhagic cerebral infarction only 3 months after the onset of the disease.