Patients with neuromuscular paralysis develop respiratory failure, mainly type II expiratory failure, with blood gases characterized by a decrease in arterial partial pressure of oxygen and an increase in partial pressure of carbon dioxide. Neuromuscular paralysis may be caused by craniocerebral trauma, phenobarbital and other sedative poisoning, neuromuscular paralysis involving the respiratory muscles will cause respiratory weakness, paralysis, decreased respiratory drive, resulting in hypoxia and carbon dioxide retention, i.e., pulmonary ventilation dysfunction, occurring type II respiratory failure. Type II respiratory failure is mainly manifested in the resting state of normal respiration, the partial pressure of oxygen at sea level <60mmHg, and the partial pressure of carbon dioxide >50mmHg. And due to neuromuscular paralysis for purely insufficient ventilation caused by the expiratory failure, generally hypoxemia and hypercapnia degree of parallel. If the patient is already on oxygen, the blood gas analysis result may also be an oxygen partial pressure of >60 mmHg, while the carbon dioxide partial pressure remains above normal. Different individual conditions in patients with neuromuscular palsy may lead to different blood gas analysis results, but overall it is a type II expiratory failure. It is recommended to analyze the patient according to his/her actual condition.