It refers to the chronic partial or complete obstruction of the portal vein or intrahepatic branches of the portal vein, resulting in obstruction of portal blood flow and causing increased portal pressure. To reduce portal hypertension, varices may occur in the superficial abdominal wall veins located around the umbilicus after the formation of collateral circulation around the portal vein or recanalization of the obstruction. In hepatic portal hypertension, the main manifestations are portal hypertension and secondary rupture of esophagogastric fundic varices and/or concomitant portal hypertensive gastropathy. Patients may have recurrent vomiting of blood and tarry stools with mild to moderate splenomegaly and hypersplenism; therefore, the liver function of such patients is good, so ascites, jaundice and hepatic encephalopathy rarely occur. Occasionally, spongy degenerative collateral vessels may compress the superficial veins of the abdominal wall around the umbilicus of the common bile duct. For patients with recurrent upper gastrointestinal bleeding, mild or moderate splenomegaly, and basically normal liver function, the possibility of CTPV should be thought of. Physical examination: Serum antinuclear antibodies, lupus anticoagulant, C protein, S protein and antithrombin III are normal, as are the international normalized ratio (INR) and activated partial thromboplastin time. However, myocardial phospholipid antibodies IgM and IgG concentrations were high, and liver function tests were normal except for albumin, serum antinuclear antibodies, lupus anticoagulant, C protein, S protein and antithrombin, and international normalized ratio (INR) and activated partial thromboplastin. 2, abdominal ultrasound: normal portal vein structure disappears, replaced by irregular curved vascular shadow, or honeycomb, in which blood flow is seen, and the direction of blood flow is irregular; the thickening of the vessel wall is echogenically enhanced, and intravascular thrombus can be seen. ueno classifies CTPV into 3 types according to the color Doppler imaging performance: type I shows the normal structure of portal vein is unclear, only the portal vein area shows a honeycomb structure, primary CTPV is of this type; type II shows that the main trunk of portal vein can be shown, but the inner part is filled with embolic material and side branch veins can be seen around it; type III shows the presence of mass echogenicity near portal vein and the formation of side branch veins due to compression of portal vein. Type II and III are secondary CTPV manifestations. 3.CT of abdomen: the direction of blood flow is irregular, and intravascular thrombus can be seen. (1) The structure of portal vein travel area is disturbed, the normal portal vein system structure disappears, and the soft tissue mesh-like structure formed by entangled side branch veins is seen in the direction of portal vein travel, and the demarcation between them is unclear, and the portal vein is obviously strengthened and intertwined into a network, sinusoidal gap-like or tube-like soft tissue structure after enhancement scan, and the thin strip-like hyperdensity shadow around the portal vein is seen in the portal part of the liver. (2) Abnormal perfusion of the liver parenchyma, in the arterial phase, the contrast agent accumulates in the peripheral part of the liver parenchyma, forming a high-density band shadow, and sometimes the proximal dilated arterial shadow can be seen, while in the portal phase the whole liver shows a uniform isointense shadow.