Many solid liver tumors present with internal bleeding, including hepatocellular adenoma, hepatocellular carcinoma, and some neuroendocrine liver metastases, and the diagnosis of bleeding is very important for treatment and prognosis determination. It is difficult to distinguish hemorrhage from solid lesions in US and CT, and MRI has a greater advantage. MRI performance] On MRI, the performance is related to the different periods of hemorrhage decomposition products. Acute hemorrhage (within 2-3 days) has a fluid signal consistent with intracellular deoxygenated hemoglobin, homogeneous signal of accumulated fluid, T1WI low signal and obvious T2WI low signal. Subacute hemorrhage (3-5 days) shows fluid signal with intracellular methemoglobin, and fluid aggregates show medium-high signal T1WI and low signal T2WI images. Late hemorrhage may present as central zone T1WI high signal (intracellular methemoglobin) T1WI and T2WI borderline low signal with iron-containing hemoglobin. Solid liver lesions usually show more pronounced mixed signal on T2WI and varying degrees of enhancement after injection of enhancer. A, T2WI fat suppression: a large mixed-signal HCC in a non-cirrhotic liver with no hemorrhage; B, T1WI isochronous image: short T1 central high signal with typical methemoglobin; C, most of the hematoma is unreinforced during the enhancement phase, while the HCC shows significant inhomogeneous enhancement; D, delayed phase: the hematoma is unreinforced, while the HCC shows multiple intratumoral segregation-like intensification. Differential diagnosis】 Hepatocellular adenoma and hepatocellular carcinoma may also show T1WI high signal in some high fat lesions.