Cerebral infarction often has various aura in the minutes, hours or days before the onset, recognizing and being alert to the occurrence of these aura and taking early measures can greatly reduce the incidence of cerebral infarction! Mortality! Degree of disability! Common aura of cerebral infarction include: 1. sudden blindness or blurred vision in one eye, which is rapidly relieved within a short period of time, mostly suggesting internal carotid artery thrombosis; 2. episodic vertigo, tinnitus, accompanied by nausea and vomiting, and unstable walking, suggesting insufficient blood supply to the vertebrobasilar system; 3. episodic slurred speech, hard and numb tongue, and choking cough; 4. episodic hemianesthesia and weakness, and drooling; 5. transient black haze 6.Sudden inability to name objects, urinary incontinence; 7.Sudden inability to recognize colors or partial blindness, memory loss; 8.Change in the nature of the original headache, from paroxysmal attacks to persistent unremitting, severe headache accompanied by nausea and vomiting; 9.Change in mood, spirit and personality. 10.The potential risk factors of cerebral infarction aura are hypertension, hyperlipidemia, history of diabetes, history of heart disease, history of atherosclerosis, long-term smoking, alcoholism and family genetic history, etc.; 11.The recent risk factors of cerebral infarction aura are: great joy and grief, overexertion, fall and trauma, sudden change of climate, sudden change of living habits, etc. The above aura can be temporary or recurrent, or even gradually aggravated. If the aura appears, you should try to maintain emotional stability, avoid excessive tension, rest in place, go to the hospital immediately after the condition stabilizes, systematically perform imaging, blood rheology, lipid, blood sugar, electrocardiogram, transcranial Doppler (TCD) and other examinations, and make the diagnosis as early as possible according to the examination results. Prompt and effective treatment should be given for transient cerebral ischemic attackers. Etiological treatment, such as hypertension, hyperlipidemia, diabetes, heart disease, etc. should be given effective treatment to correct: such as vasodilator drug application, improvement of blood circulation, anticoagulation therapy, cerebral metabolic activator, microsurgery treatment, etc. In short, to convert transient cerebral ischemia to the normal direction, the occurrence of irreversible cerebral infarction can be blocked.