The patient was a female, 51 years old. She came to our outpatient clinic in April 2014 due to the discovery of yellowish-white streaks on her lower back for 2 months. The patient came to our clinic 2 months ago after her family suddenly noticed several palpable yellow-white stripes on her lower back, slightly elevated on the skin, without any conscious symptoms. He denied any history of significant weight gain or loss, any history of trauma to the skin lesions, any history of systematic glucocorticoid treatment, any family history of hereditary disease, and no family history of similar disease. Physical examination: No significant abnormalities were found in the systemic examination of the whole body. Dermatologic examination: several transverse palpable yellow-white streaks on the lower back, slightly elevated, without tenderness or induration. Laboratory tests: Blood, urine, feces, liver and kidney functions were not significantly abnormal. The epidermis was basically normal; the dermal collagen fibers were mildly hyperplastic and hypertrophic, and the dermal reticular layer had fewer elastic fibers, with localized clustered distribution of fragmented elastic fibers. Burket et al. first reported linearfocalelastosis (LFE) in 1989, in which three patients, all over 60 years old, presented with asymptomatic, palpable yellow stripes on their lower and middle back. At that time, it was also called “elastoticstriae”. However, more scholars tend to use the term “linear limited elastosis (LFE)”, which is considered to be an independent disease compared to the swelling striae. Although there are few reports on this disease at home and abroad, only one case of linear restrictive elastic tissue degeneration in children was reported by Guangcai Xiang et al. in China, and one misdiagnosed case was reported by Zhiguo Li [5]. However, we believe that its clinical appearance is not uncommon, only that sometimes it is ignored by patients or lack of awareness by clinicians, or even misdiagnosed as swelling striae. The typical clinical manifestation of the disease is a slightly elevated, palpable, yellow stripe across the lower back with no conscious symptoms, usually in elderly men, but also in women and children. The pathogenesis of the disease is also uncertain, and ChoiSW et al. suggested that it is due to excessive degeneration/proliferation of elastic fibers. Although controversial, some scholars believe that LFE is associated with swollen lines (striaeDistensae (SD)) due to excessive proliferation of elastic fibers in late SD, i.e., keloidal repairprocess (KRP) of elastic fibers. This hypothesis comes from their finding that some patients had both SD and LFE, especially JeongS et al. reported one patient with both SD and LFE in the posterior dorsum, and LFE occurred after SD, which seems to support KRP as one of the pathogenesis of LFE, but the pathogenesis cannot be conclusively determined because the disease has been reported rarely and mostly in individual cases. Verhoeff-vanGieson staining confirmed the origin of these materials from degenerated or degenerated elastic fibers. Choi SW et al. and SeoJK et al. reported two cases at the same time with varying durations of disease. Through these cases, they found that the histopathological manifestations of the disease can vary between early and late stages, with the early stage showing degenerative degeneration of elastic fibers with reduced numbers, and the late stage showing abnormal proliferation of elastic fibers with increased numbers, deposited in fine waves or fragments between normal or mildly proliferated collagen fibers. In addition, AdişenE et al. also reported a patient with a 3-month history of the disease with histopathological manifestations of degeneration of elastic tissue, with fragmented deposition between mildly hyperplastic collagen fibers and a decrease in their number. Therefore, we speculate that the histopathological manifestation of this disease is a process of gradual change with the course of the disease, and the cases reported by some scholars in the past may only be the manifestation of a specific period of the disease, but the common histopathological manifestations are normal epidermis, normal or mild hyperplasia and hypertrophy of dermal collagen fibers, degeneration and degeneration of elastic fibers, and in the advanced stage, abnormal proliferation of elastic fibers deposited between collagen fiber bundles. The clinical manifestation of this patient was a typical yellow-white stripe palpable on the lower back, and the histopathology was normal epidermis, mild hyperplasia and hypertrophy of dermal collagen fibers, localized degenerative elastic fibers in fragmented clusters between collagen fibers, and reduced elastic fibers in the dermal reticular layer, which may be related to the short duration of the patient’s disease. The disease needs to be clinically differentiated mainly from swelling lines. The basic damage is a palpable yellowish-white stripe, often without sudden weight gain, history of glucocorticoid use, pregnancy, etc. The histopathology is characterized by degeneration and degeneration of the dermal elastic tissue, with a cluster-like distribution of amorphous material, decreasing in number in the early stages and increasing in the later stages. The skin histopathology is mainly characterized by epidermal atrophy, dermal thinning, degeneration and separation of collagen fibers, and scarce, curled or lumpy elastic fibers in the reticular layer.