Vestibular ataxia is caused by damage to the vestibular system and is predominantly a balance disorder. It is characterized by balance disturbances at rest and during movement. It has similarities with cerebellar ataxia, such as wide base of both feet when standing, unsteadiness, leaning to the side or back, and skewing when walking. However, they are generally distinguished by the presence of vertigo, nystagmus and abnormal vestibular function tests. Depending on the location of the lesion, the disease can be divided into peripheral vestibular ataxia, which is caused by lesions in the vestibule of the inner ear to the vestibular nerve, and central vestibular ataxia, which is caused by lesions in the vestibular nucleus and its center. The disease is not yet specific and can be alleviated symptomatically: 1. viable optic thalamus destruction; 2. training, physical and assistive walking devices are beneficial, carry out genetics; 2. levodopa can alleviate extrapyramidal system such as ankylosis, toxic lentil base or cytidyl phosphorylcholine promotes acetylcholine synthesis; chlorambucil can reduce spasticity, amantadine can improve ataxia, ataxia with myoclonus preferred to hushnitropin; ATP, coenzyme A ATP, coenzyme A, inosine, and vitamin B neurotrophic drugs are tried.