Vestibular ataxia is caused by damage to the vestibular system and is predominantly a balance disorder. It is characterized by balance disturbances at rest and during movement. It has similarities with cerebellar ataxia, such as wide base of both feet when standing, unsteadiness, leaning to the side or back, and skewing when walking. However, they are generally distinguished by the presence of significant vertigo, nystagmus and abnormal vestibular function tests. Vestibular ataxia is a disorder of spatial orientation caused by vestibular lesions, mainly balance disorders, manifesting unsteadiness in standing, leaning to the side of the disease when walking, walking in a straight line, changing head position, and ataxic movements of the limbs; often accompanied by vertigo, vomiting and nystagmus. The vestibular function of the inner ear is diminished or absent with the warming (hot and cold water) test or rotation test. The closer the lesion is to the inner ear vagus, the more ataxic it is. The disease is not yet specific, symptomatic relief can be achieved: 1, levodopa can relieve tonic and other extrapyramidal system, toxic lentil base or cytidylcholine to promote acetylcholine synthesis; chlorambucil can reduce spasticity, amantadine can improve ataxia, ataxia with myoclonus preferred to hush nitroprusside; ATP, coenzyme A, inosine and vitamin B and other neurotrophic drugs trial; 2, feasible optic thalamus destruction; 3, training, physical and assistive walking devices are beneficial and genetic.