Luminal infarction A luminal infarction is a type of cerebral infarction that accounts for approximately 20% of cerebral infarctions and is relatively mild. The disease is caused by ischemic microinfarction due to wall lesions and luminal occlusion of small deep penetrating arteries in the brain tissue. The ischemic necrotic and liquefied brain tissue is removed by phagocytes, resulting in a cavity in the lesion, hence the term “cerebral lacunar infarction” (lacunar infarction). The lesion characteristics of lacunar infarction: the lesions are small, ranging from 2 to 20 mm, mostly 2 to 4 mm, and mostly located in the deep brain tissue (mostly in the basal ganglia). Clinical characteristics of lacunar infarction: the severity varies widely. Most of them have no obvious symptoms, and about 3/4 of the patients have no symptoms of focal neurological damage, or only mild inattention, memory loss, mild headache and dizziness, vertigo, unresponsiveness, mild numbness of upper and lower extremities, unstable walking, weakness of clenching fist, trembling of hands, feet or neck, and hypoesthesia or abnormality. And sometimes there are recurrent attacks. Some of the multiple cerebral cavernous infarcts can affect brain function, leading to progressive mental decline and finally cerebrovascular dementia without coma and other impairment of consciousness. The diagnosis of the disease is mainly based on CT or MRI examinations, and the notion that lacunar infarcts do not require treatment or are untreatable is inappropriate. It is proved that the symptoms can be significantly reduced or eliminated and the whole brain blood supply improved after effective treatment (Chinese medicine compound is more effective), but the luminal lesions will not disappear (no effect on normal function).